{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,7,2]],"date-time":"2026-07-02T20:28:55Z","timestamp":1783024135661,"version":"3.54.6"},"reference-count":13,"publisher":"Ovid Technologies (Wolters Kluwer Health)","issue":"8","funder":[{"name":"Beijing Anzhen Hospital High Level Research Funding","award":["2025AZB6013"],"award-info":[{"award-number":["2025AZB6013"]}]}],"content-domain":{"domain":["lww.com","ovid.com"],"crossmark-restriction":true},"short-container-title":[],"published-print":{"date-parts":[[2026,8]]},"abstract":"<jats:p>\n                    Inflammation-associated coronary artery disease (I-CAD) is a recognized yet underdiagnosed cause of refractory in-stent restenosis. Conventional imaging modalities often lack sensitivity for detecting focal inflammatory activity. A 74-year-old woman with recurrent ISR was admitted to our institution. Dual-tracer \u00b9\u2078F-FAPI and \u00b9\u2078F-FDG PET\/CT revealed locally increased radiotracer uptake within the left anterior descending artery (LAD) stent segment, with more intense uptake on\n                    <jats:sup>18<\/jats:sup>\n                    F-FAPI than on\n                    <jats:sup>18<\/jats:sup>\n                    F-FDG. We present the dual-tracer PET\/CT findings in this patient with recurrent in-stent restenosis, suggesting active peri-stent fibroblast-related remodeling and an inflammation-associated mechanism. The patient was subsequently managed with anti-inflammatory therapy rather than repeated revascularization.\n                  <\/jats:p>","DOI":"10.1097\/rlu.0000000000006563","type":"journal-article","created":{"date-parts":[[2026,6,8]],"date-time":"2026-06-08T13:01:04Z","timestamp":1780923664000},"page":"742-743","update-policy":"https:\/\/doi.org\/10.1097\/lww.0000000000001000","source":"Crossref","is-referenced-by-count":0,"title":["18F-FAPI Outperforms 18F-FDG PET\/CT in Detecting Focal Inflammation as a Cause of Refractory In-Stent Restenosis"],"prefix":"10.1097","volume":"51","author":[{"given":"Hangyu","family":"Xie","sequence":"first","affiliation":[{"name":"Department of Nuclear Medicine, Beijing Anzhen Hospital, Capital Medical University, Beijing Institute of Heart, Lung and Blood Vessel Diseases"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Xu","family":"Han","sequence":"additional","affiliation":[{"name":"Department of Nuclear Medicine, Beijing Anzhen Hospital, Capital Medical University, Beijing Institute of Heart, Lung and Blood Vessel Diseases"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Hongzhi","family":"Mi","sequence":"additional","affiliation":[{"name":"Department of Nuclear Medicine, Beijing Anzhen Hospital, Capital Medical University, Beijing Institute of Heart, Lung and Blood Vessel Diseases"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Haiyan","family":"Qian","sequence":"additional","affiliation":[{"name":"Department of Cardiology, Beijing AnZhen Hospital, Capital Medical University, National Clinical Research Center for Cardiovascular Diseases, Office of Beijing Cardiovascular Diseases Prevention, Beijing, China"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Wei","family":"Dong","sequence":"additional","affiliation":[{"name":"Department of Nuclear Medicine, Beijing Anzhen Hospital, Capital Medical University, Beijing Institute of Heart, Lung and Blood Vessel 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