{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,9,18]],"date-time":"2026-09-18T04:23:20Z","timestamp":1789705400470,"version":"4.0.1"},"reference-count":40,"publisher":"Microbiology Society","issue":"6","content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":[],"published-print":{"date-parts":[[2014,6,1]]},"abstract":"<jats:p>\n                    The cucumber mosaic virus (CMV) 2b silencing suppressor protein allows the virus to overcome resistance to replication and local movement in inoculated leaves of plants treated with salicylic acid (SA), a resistance-inducing plant hormone. In\n                    <jats:italic>Arabidopsis thaliana<\/jats:italic>\n                    plants systemically infected with CMV, the 2b protein also primes the induction of SA biosynthesis during this compatible interaction. We found that CMV infection of susceptible tobacco (\n                    <jats:italic>Nicotiana tabacum<\/jats:italic>\n                    ) also induced SA accumulation. Utilization of mutant 2b proteins expressed during infection of tobacco showed that the N- and C-terminal domains, which had previously been implicated in regulation of symptom induction, were both required for subversion of SA-induced resistance, while all mutants tested except those affecting the putative phosphorylation domain had lost the ability to prime SA accumulation and expression of the SA-induced marker gene\n                    <jats:italic>PR-1<\/jats:italic>\n                    .\n                  <\/jats:p>","DOI":"10.1099\/vir.0.063461-0","type":"journal-article","created":{"date-parts":[[2014,3,14]],"date-time":"2014-03-14T20:15:13Z","timestamp":1394828113000},"page":"1408-1413","source":"Crossref","is-referenced-by-count":51,"title":["Domains of the cucumber mosaic virus 2b silencing suppressor protein affecting inhibition of salicylic acid-induced resistance and priming of salicylic acid accumulation during 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