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Because no known fungicides or cotton cultivars provide sufficient protection against this pathogen,\n                    <jats:styled-content style=\"fixed-case\">\n                      <jats:italic>V<\/jats:italic>\n                    <\/jats:styled-content>\n                    <jats:italic>.\u2009dahliae<\/jats:italic>\n                    causes major crop yield losses. Here, an isolated cotton endophytic bacterium, designated\n                    <jats:styled-content style=\"fixed-case\">\n                      <jats:italic>B<\/jats:italic>\n                    <\/jats:styled-content>\n                    <jats:italic>acillus amyloliquefaciens<\/jats:italic>\n                    41\n                    <jats:styled-content style=\"fixed-case\">B<\/jats:styled-content>\n                    \u20101, exhibited greater than 50% biocontrol efficacy against\n                    <jats:styled-content style=\"fixed-case\">\n                      <jats:italic>V<\/jats:italic>\n                    <\/jats:styled-content>\n                    <jats:italic>.\u2009dahliae<\/jats:italic>\n                    in cotton plants under greenhouse conditions. Through high\u2010performance liquid chromatography and mass analysis of the filtrate, we found that the antifungal compounds present in the strain 41\n                    <jats:styled-content style=\"fixed-case\">B<\/jats:styled-content>\n                    \u20101 culture filtrate were a series of isoforms of iturins. The purified iturins suppressed\n                    <jats:styled-content style=\"fixed-case\">\n                      <jats:italic>V<\/jats:italic>\n                    <\/jats:styled-content>\n                    <jats:italic>.\u2009dahliae<\/jats:italic>\n                    microsclerotial germination in the absence or presence of cotton. Treatment with the iturins induced reactive oxygen species bursts,\n                    <jats:styled-content style=\"fixed-case\">H<\/jats:styled-content>\n                    og1 mitogen\u2010activated protein kinase (MAPK) activation and defects in cell wall integrity. The oxidative stress response and high\u2010osmolarity glycerol pathway contribute to iturins resistance in\n                    <jats:styled-content style=\"fixed-case\">\n                      <jats:italic>V<\/jats:italic>\n                    <\/jats:styled-content>\n                    <jats:italic>.\u2009dahliae<\/jats:italic>\n                    . In contrast, the\n                    <jats:styled-content style=\"fixed-case\">S<\/jats:styled-content>\n                    lt2\n                    <jats:styled-content style=\"fixed-case\">MAPK<\/jats:styled-content>\n                    pathway may be involved in iturins sensitivity in this fungus. In addition to antagonism, iturins could induce plant defence responses as activators and mediate pathogen\u2010associated molecular pattern\u2010triggered immunity. These findings suggest that iturins may affect fungal signalling pathways and mediate plant defence responses against\n                    <jats:styled-content style=\"fixed-case\">\n                      <jats:italic>V<\/jats:italic>\n                    <\/jats:styled-content>\n                    <jats:italic>.\u2009dahliae<\/jats:italic>\n                    .\n                  <\/jats:p>","DOI":"10.1111\/1462-2920.12538","type":"journal-article","created":{"date-parts":[[2014,6,16]],"date-time":"2014-06-16T23:39:36Z","timestamp":1402961976000},"page":"1166-1188","update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":168,"title":["The bacterial lipopeptide iturins induce\n                    <scp>\n                      <i>V<\/i>\n                    <\/scp>\n                    <i>erticillium dahliae<\/i>\n                    cell death by affecting fungal signalling pathways and mediate plant defence responses involved in 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