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This review systematically elucidates the critical role of T cell immunosenescence in the pathogenesis of common inflammatory skin diseases, including psoriasis, atopic dermatitis, rosacea, and seborrheic dermatitis. Senescent T cells drive the production of a disease\u2010specific SASP via internally dysregulated signaling networks such as NF\u2010\u03baB, JAK\u2010STAT, p38 MAPK, and PI3K\u2010Akt\u2010mTOR pathways, thereby shaping and sustaining a chronic cutaneous inflammatory microenvironment that promotes disease chronicity and recurrence. Furthermore, this review summarizes current therapeutic strategies targeting these senescence\u2010associated pathways and SASP components, discussing both biological agents and small molecule inhibitors. 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