{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,9,26]],"date-time":"2026-09-26T17:04:41Z","timestamp":1790442281888,"version":"4.1.0"},"reference-count":37,"publisher":"Wiley","issue":"3","license":[{"start":{"date-parts":[[2013,10,24]],"date-time":"2013-10-24T00:00:00Z","timestamp":1382572800000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Alcoholism Clin &amp;amp; Exp Res"],"published-print":{"date-parts":[[2014,3]]},"abstract":"<jats:sec><jats:title>Background<\/jats:title><jats:p>Prenatal alcohol exposure (<jats:styled-content style=\"fixed-case\">PAE<\/jats:styled-content>) is a leading cause of significant neurobehavioral and neurocognitive deficits. Its potential consequences for eating behaviors, nutritional status, and other nutritional issues in childhood have received little attention.<\/jats:p><\/jats:sec><jats:sec><jats:title>Methods<\/jats:title><jats:p>Nineteen children (11 boys, 8 girls) of mean age 9.6\u00a0years, referred for fetal alcohol spectrum disorder (<jats:styled-content style=\"fixed-case\">FASD<\/jats:styled-content>) screening and assessment, were analyzed with physical exams and caregiver questionnaires to identify possible abnormalities in food and eating behaviors. Fourteen children contributed 24\u2010hour diet recalls and were assessed for nutritional status.<\/jats:p><\/jats:sec><jats:sec><jats:title>Results<\/jats:title><jats:p>Seventy\u2010nine percent of participants were diagnosed with <jats:styled-content style=\"fixed-case\">FASD<\/jats:styled-content> and 63.2% had confirmed <jats:styled-content style=\"fixed-case\">PAE<\/jats:styled-content>. Fifty percent of girls were overweight or obese, whereas 37% of boys had reduced stature, weight, or body mass index for their age. Recurring feeding problems included constant snacking (36.8%), lack of satiety (26.3%), and picky eating\/poor appetite (31.6%). None had oral feeding problems. Constipation was common (26.3%). Macronutrient intakes were largely normal, but sugar consumption was excessive (140 to 190% of recommendations) in 57% of subjects. Vitamin <jats:styled-content style=\"fixed-case\">A<\/jats:styled-content> intake exceeded the upper limit for 64% of participants, whereas \u226550% had intakes &lt;80% of recommended daily allowances (<jats:styled-content style=\"fixed-case\">RDA<\/jats:styled-content>s) for choline, vitamin <jats:styled-content style=\"fixed-case\">E<\/jats:styled-content>, potassium, \u03b2\u2010carotene, and essential fatty acids; 100% had vitamin <jats:styled-content style=\"fixed-case\">D<\/jats:styled-content> intakes &lt;80% of the <jats:styled-content style=\"fixed-case\">RDA<\/jats:styled-content>.<\/jats:p><\/jats:sec><jats:sec><jats:title>Conclusions<\/jats:title><jats:p>PAE may be associated with altered acquisition and distribution of body mass with increasing age. Disordered eating was common. The increased feeding behaviors surrounding lack of satiety suggest that self\u2010regulation may be altered. Constipation could reflect low dietary fiber or altered gastrointestinal function. These exploratory data suggest that children with <jats:styled-content style=\"fixed-case\">PAE<\/jats:styled-content> may be at risk for nutritional deficiencies, which are influenced by inappropriate food preferences, disordered eating patterns, medication use, and the stressful dynamics surrounding food preparation and mealtime.<\/jats:p><\/jats:sec>","DOI":"10.1111\/acer.12284","type":"journal-article","created":{"date-parts":[[2013,10,24]],"date-time":"2013-10-24T16:36:57Z","timestamp":1382632617000},"page":"871-878","source":"Crossref","is-referenced-by-count":54,"title":["Inappropriate Feeding Behaviors and Dietary Intakes in Children with Fetal Alcohol Spectrum Disorder or Probable Prenatal Alcohol Exposure"],"prefix":"10.1111","volume":"38","author":[{"given":"Rachel L.","family":"Werts","sequence":"first","affiliation":[{"name":"Waisman Center University of Wisconsin School of Medicine and Public Health Madison Wisconsin"},{"name":"Department of Nutritional Sciences University of Wisconsin\u2010Madison Madison Wisconsin"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Sandra C.","family":"Van Calcar","sequence":"additional","affiliation":[{"name":"Waisman Center University of Wisconsin School of Medicine and Public Health Madison Wisconsin"},{"name":"Department of Pediatrics University of Wisconsin School of Medicine and Public Health Madison Wisconsin"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"David S.","family":"Wargowski","sequence":"additional","affiliation":[{"name":"Waisman Center University of Wisconsin School of Medicine and Public Health Madison Wisconsin"},{"name":"Department of Pediatrics University of Wisconsin School of Medicine and Public Health Madison Wisconsin"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Susan M.","family":"Smith","sequence":"additional","affiliation":[{"name":"Waisman Center University of Wisconsin School of Medicine and Public Health Madison Wisconsin"},{"name":"Department of Nutritional Sciences University of Wisconsin\u2010Madison Madison Wisconsin"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"311","published-online":{"date-parts":[[2013,10,24]]},"reference":[{"key":"e_1_2_6_2_1","doi-asserted-by":"publisher","DOI":"10.3945\/jn.109.118539"},{"key":"e_1_2_6_3_1","doi-asserted-by":"publisher","DOI":"10.1542\/peds.101.S2.539"},{"key":"e_1_2_6_4_1","unstructured":"ChesterDN GoldmanJD AhujaJK MoshfeghAJ(2011)Dietary Intakes of Choline: What We Eat in America NHANES 2007\u20132008. 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