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The association between <jats:styled-content style=\"fixed-case\">BZDR<\/jats:styled-content> use and <jats:styled-content style=\"fixed-case\">AD<\/jats:styled-content> was assessed using conditional logistic regression with 5\u2010year lag time between exposure and outcome.<\/jats:p><\/jats:sec><jats:sec><jats:title>Results<\/jats:title><jats:p>Benzodiazepine and related drug use was associated with modestly increased risk of <jats:styled-content style=\"fixed-case\">AD<\/jats:styled-content> (adjusted <jats:styled-content style=\"fixed-case\">OR<\/jats:styled-content> 1.06, 95% <jats:styled-content style=\"fixed-case\">CI<\/jats:styled-content> 1.04\u20131.08). A dose\u2013response relationship was observed with both cumulative consumption and duration. Adjustment for other psychotropics removed the cumulative dose\u2013response relationship by attenuating the <jats:styled-content style=\"fixed-case\">OR<\/jats:styled-content>s in the highest dose category.<\/jats:p><\/jats:sec><jats:sec><jats:title>Conclusion<\/jats:title><jats:p>Benzodiazepine and related drug use in general was associated with modestly increased risk of <jats:styled-content style=\"fixed-case\">AD<\/jats:styled-content>. No major differences were observed between different subcategories of <jats:styled-content style=\"fixed-case\">BZDR<\/jats:styled-content>s (i.e. benzodiazepines, Z drugs, short\u2010\/medium\u2010acting or long\u2010acting <jats:styled-content style=\"fixed-case\">BZDR<\/jats:styled-content>s). 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