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Participants were emerging adults (N\u2009=\u2009218\u2013268 [52\u201356% female], age 18\u201320).<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Measurements<\/jats:title>\n                    <jats:p>Participants completed an alcohol cue approach\u2010avoidance task while their electroencephalogram (EEG) was recorded. Behavioral measures (response time) indexed the strength of cue\u2010activated approach vs. avoidance tendency. Cue\u2010locked event\u2010related potentials provided EEG\u2010based neural measures of motivated attention (P3 amplitude) and approach\u2010avoidance conflict (N450 amplitude). From EMA, measures of alcohol consumption dynamics (as indexed by estimated blood alcohol concentration [eBAC], g\/dL) during real\u2010world drinking episodes were obtained, as were measures of alcohol craving (7\u2010point visual analogue scale) dynamics during and outside these episodes.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Findings<\/jats:title>\n                    <jats:p>Different approach\u2010avoidance task\u2010derived behavioral and neural measures rank\u2010ordered participants differently.<\/jats:p>\n                    <jats:p>\n                      Participants who\n                      <jats:italic>approached<\/jats:italic>\n                      alcohol cues more rapidly in lab subsequently showed steeper increases in craving (\u2206B\u2009\u00b1\u2009standard error [SE]\u2009=\u20091.042\u2009\u00b1\u20090.499 point\/hr), and eBAC (\u2206B\u2009\u00b1\u2009SE\u2009=\u20090.046\u2009\u00b1\u20090.017\u2009g\/dl\/hr), during real\u2010world drinking episodes. Participants who\n                      <jats:italic>avoided<\/jats:italic>\n                      alcohol cues more slowly in lab also showed steeper increases in eBAC (\u2206B\u2009\u00b1\u2009SE\u2009=\u20090.056\u2009\u00b1\u20090.017\u2009g\/dl\/hr).\n                    <\/jats:p>\n                    <jats:p>\n                      Participants with larger P3 during alcohol cue\n                      <jats:italic>approach<\/jats:italic>\n                      in lab subsequently showed steeper increases in eBAC (\u2206B\u2009\u00b1\u2009SE\u2009=\u20090.048\u2009\u00b1\u20090.017\u2009g\/dl\/hr), as did those with smaller P3 during alcohol cue\n                      <jats:italic>avoidance<\/jats:italic>\n                      (\u2206B\u2009\u00b1\u2009SE\u2009=\u20090.025\u2009\u00b1\u20090.017\u2009g\/dl\/hr).\n                    <\/jats:p>\n                    <jats:p>\n                      Participants with smaller N450 during alcohol cue\n                      <jats:italic>approach<\/jats:italic>\n                      in lab subsequently showed steeper increases in craving during drinking episodes (\u2206B\u2009\u00b1\u2009SE\u2009=\u20091.465\u2009\u00b1\u20090.607 point\/hr).\n                    <\/jats:p>\n                    <jats:p>Tests examining lab\u2010based neurobehavioral measures as predictors of craving dynamics during nondrinking moments, such as following incidental cue exposure, generally were inconclusive.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Conclusions<\/jats:title>\n                    <jats:p>Incentive salience toward alcohol may influence alcohol seeking (including craving) and alcohol consumption through distinct behavioral risk pathways in different people.<\/jats:p>\n                  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