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Here, we systematically evaluated the literature on the prevalence of thyroid autoimmunity in <jats:styled-content style=\"fixed-case\">CSU<\/jats:styled-content> and vice versa. There is a strong link between <jats:styled-content style=\"fixed-case\">CSU<\/jats:styled-content> and elevated levels of IgG antithyroid autoantibodies (<jats:styled-content style=\"fixed-case\">AA<\/jats:styled-content>bs), with most of a large number of studies reporting rates of \u226510%. Levels of IgG against thyroid peroxidase (<jats:styled-content style=\"fixed-case\">TPO<\/jats:styled-content>) are more often elevated in <jats:styled-content style=\"fixed-case\">CSU<\/jats:styled-content> than those of other IgG antithyroid <jats:styled-content style=\"fixed-case\">AA<\/jats:styled-content>bs (strong evidence). Levels of IgG antithyroid <jats:styled-content style=\"fixed-case\">AA<\/jats:styled-content>bs are more often elevated in adult patients with <jats:styled-content style=\"fixed-case\">CSU<\/jats:styled-content> than in children (strong evidence). Patients with <jats:styled-content style=\"fixed-case\">CSU<\/jats:styled-content> exhibit significantly higher levels of IgG antithyroid <jats:styled-content style=\"fixed-case\">AA<\/jats:styled-content>bs (strong evidence) and IgE\u2010anti\u2010<jats:styled-content style=\"fixed-case\">TPO<\/jats:styled-content> (weak evidence) than controls. Elevated IgG antithyroid <jats:styled-content style=\"fixed-case\">AA<\/jats:styled-content>bs in <jats:styled-content style=\"fixed-case\">CSU<\/jats:styled-content> are linked to the use of glucocorticoids (weak evidence) but not to disease duration or severity\/activity, gender, age, or <jats:styled-content style=\"fixed-case\">ASST<\/jats:styled-content> response (inconsistent evidence). Thyroid dysfunction rates are increased in patients with <jats:styled-content style=\"fixed-case\">CSU<\/jats:styled-content> (strong evidence). Hypothyroidism and Hashimoto's thyroiditis are more common than hyperthyroidism and Graves\u2019 disease (strong evidence). Thyroid dysfunction is more common in adult patients with <jats:styled-content style=\"fixed-case\">CSU<\/jats:styled-content> than in children (strong evidence) and in female than in male patients with <jats:styled-content style=\"fixed-case\">CSU<\/jats:styled-content> (weak evidence). Urticaria including <jats:styled-content style=\"fixed-case\">CSU<\/jats:styled-content> is more prevalent in patients with thyroid autoimmunity than in controls (weak evidence). <jats:styled-content style=\"fixed-case\">CSU<\/jats:styled-content> can improve in response to treatment with levothyroxine or other thyroid drugs (strong evidence). Pathogenic mechanisms in <jats:styled-content style=\"fixed-case\">CSU<\/jats:styled-content> patients with thyroid autoimmunity may include IgE against autoantigens, immune complexes, and complement.<\/jats:p>","DOI":"10.1111\/all.13182","type":"journal-article","created":{"date-parts":[[2017,4,13]],"date-time":"2017-04-13T18:37:34Z","timestamp":1492108654000},"page":"1440-1460","source":"Crossref","is-referenced-by-count":167,"title":["Comorbidity of chronic spontaneous urticaria and autoimmune thyroid diseases: A systematic review"],"prefix":"10.1111","volume":"72","author":[{"given":"P.","family":"Kolkhir","sequence":"first","affiliation":[{"name":"Department of Dermatology and Venereology I.M. 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