{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,9,28]],"date-time":"2026-09-28T00:02:14Z","timestamp":1790553734447,"version":"4.1.0"},"reference-count":24,"publisher":"Wiley","issue":"3","license":[{"start":{"date-parts":[[2017,12,4]],"date-time":"2017-12-04T00:00:00Z","timestamp":1512345600000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"funder":[{"name":"Torbj\u00f6rn Jebner Memorial Foundation"},{"name":"Swedish government","award":["ALFGBG-531951"],"award-info":[{"award-number":["ALFGBG-531951"]}]}],"content-domain":{"domain":["onlinelibrary.wiley.com"],"crossmark-restriction":true},"short-container-title":["Acta Paediatrica"],"published-print":{"date-parts":[[2018,3]]},"abstract":"Abstract<\/jats:title>\n \n Aim<\/jats:title>\n \n This was the 12th population\u2010based study to explore the epidemiology of cerebral palsy (\n CP<\/jats:styled-content>\n ) in western Sweden.\n <\/jats:p>\n <\/jats:sec>\n \n Methods<\/jats:title>\n From 2007 to 2010, there were 104\u00a0713 live births in the area. We analysed the birth characteristics, aetiology and neuroimaging findings, calculated the prevalence and compared the results with previous study cohorts.<\/jats:p>\n <\/jats:sec>\n \n Results<\/jats:title>\n \n Cerebral palsy was found in 205 children, corresponding to a crude prevalence of 1.96 per 1000 live births. The gestational age\u2010specific prevalence for <28 gestational weeks was 59.0 per 1000 live births, 45.7 for 28\u201331\u00a0weeks, 6.0 for 32\u201336\u00a0weeks and 1.2 for >36\u00a0weeks. Hemiplegia accounted for 44%, diplegia for 34%, tetraplegia for 5%, dyskinetic\n CP<\/jats:styled-content>\n for 12% and ataxia for 3%. Neuroimaging showed maldevelopment in 12%, white matter lesions in 49%, cortical\/subcortical lesions in 15% and basal ganglia lesions in 11%. The aetiology was considered prenatal in 38%, peri\/neonatal in 38% and remained unclassified in 24%.\n CP<\/jats:styled-content>\n due to term or near\u2010term asphyxia had decreased.\n <\/jats:p>\n <\/jats:sec>\n \n Conclusion<\/jats:title>\n \n A nonsignificant decrease in\n CP<\/jats:styled-content>\n prevalence was seen in term\u2010born children. Hemiplegia was still the most prevalent\n CP<\/jats:styled-content>\n type, while the prevalence of dyskinetic\n CP<\/jats:styled-content>\n had decreased. One in two children had white matter lesions, indicating late second\u2010 or early third\u2010trimester timing.\n <\/jats:p>\n <\/jats:sec>","DOI":"10.1111\/apa.14147","type":"journal-article","created":{"date-parts":[[2017,11,9]],"date-time":"2017-11-09T15:43:05Z","timestamp":1510242185000},"page":"462-468","update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":104,"title":["The panorama of cerebral palsy in Sweden part\n XII<\/scp>\n shows that patterns changed in the birth years 2007\u20132010"],"prefix":"10.1111","volume":"107","author":[{"given":"K","family":"Himmelmann","sequence":"first","affiliation":[{"name":"Department of Pediatrics Institute of Clinical Sciences Sahlgrenska Academy at the University of Gothenburg G\u00f6teborg Sweden"},{"name":"Regional Rehabilitation Centre Queen Silvia Children's Hospital G\u00f6teborg Sweden"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"P","family":"Uvebrant","sequence":"additional","affiliation":[{"name":"Department of Pediatrics Institute of Clinical Sciences Sahlgrenska Academy at the University of Gothenburg G\u00f6teborg Sweden"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"311","published-online":{"date-parts":[[2017,12,4]]},"reference":[{"key":"e_1_2_11_2_1","doi-asserted-by":"publisher","DOI":"10.1111\/apa.12614"},{"key":"e_1_2_11_3_1","doi-asserted-by":"publisher","DOI":"10.1111\/dmcn.12865"},{"key":"e_1_2_11_4_1","doi-asserted-by":"publisher","DOI":"10.1111\/dmcn.13001"},{"key":"e_1_2_11_5_1","doi-asserted-by":"publisher","DOI":"10.1016\/j.braindev.2016.03.007"},{"key":"e_1_2_11_6_1","doi-asserted-by":"publisher","DOI":"10.1111\/ppe.12299"},{"key":"e_1_2_11_7_1","doi-asserted-by":"publisher","DOI":"10.1001\/jamapediatrics.2016.1210"},{"key":"e_1_2_11_8_1","doi-asserted-by":"publisher","DOI":"10.1111\/dmcn.12017"},{"key":"e_1_2_11_9_1","doi-asserted-by":"publisher","DOI":"10.1111\/j.1600-0412.2011.01217.x"},{"key":"e_1_2_11_10_1","doi-asserted-by":"publisher","DOI":"10.1186\/1471-2431-8-17"},{"key":"e_1_2_11_11_1","volume-title":"Cerebral palsies: Epidemiology and causal pathways","author":"Stanley F","year":"2000"},{"key":"e_1_2_11_12_1","first-page":"8","article-title":"A report: the definition and classification of cerebral palsy April 2006","volume":"109","author":"Rosenbaum P","year":"2007","journal-title":"Dev Med Child Neurol"},{"key":"e_1_2_11_13_1","doi-asserted-by":"publisher","DOI":"10.1111\/j.1651-2227.1975.tb03820.x"},{"key":"e_1_2_11_14_1","doi-asserted-by":"publisher","DOI":"10.1111\/j.1651-2227.1996.tb14193.x"},{"key":"e_1_2_11_15_1","doi-asserted-by":"publisher","DOI":"10.1111\/j.1469-8749.2000.tb00695.x"},{"key":"e_1_2_11_16_1","doi-asserted-by":"publisher","DOI":"10.1111\/j.1651-2227.1988.tb14939.x"},{"key":"e_1_2_11_17_1","doi-asserted-by":"publisher","DOI":"10.1111\/j.1651-2227.1991.tb11945.x"},{"key":"e_1_2_11_18_1","doi-asserted-by":"publisher","DOI":"10.1016\/j.expneurol.2004.05.037"},{"key":"e_1_2_11_19_1","doi-asserted-by":"publisher","DOI":"10.1111\/dmcn.13166"},{"key":"e_1_2_11_20_1","doi-asserted-by":"publisher","DOI":"10.1016\/S0022-3476(78)80282-0"},{"key":"e_1_2_11_21_1","doi-asserted-by":"publisher","DOI":"10.1111\/j.1469-8749.1995.tb11992.x"},{"key":"e_1_2_11_22_1","volume-title":"Neonatal encephalopathy and cerebral palsy: defining the pathogenesis and pathophysiology","author":"ACOG. 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