{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,10,23]],"date-time":"2026-10-23T05:38:39Z","timestamp":1792733919106,"version":"4.5.0"},"reference-count":186,"publisher":"Wiley","issue":"11","license":[{"start":{"date-parts":[[2026,10,7]],"date-time":"2026-10-07T00:00:00Z","timestamp":1791331200000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"},{"start":{"date-parts":[[2026,10,7]],"date-time":"2026-10-07T00:00:00Z","timestamp":1791331200000},"content-version":"tdm","delay-in-days":0,"URL":"http:\/\/doi.wiley.com\/10.1002\/tdm_license_1.1"}],"content-domain":{"domain":["onlinelibrary.wiley.com"],"crossmark-restriction":true},"short-container-title":["Acta Physiologica"],"published-print":{"date-parts":[[2026,11]]},"abstract":"<jats:title>ABSTRACT<\/jats:title>\n                  <jats:p>Hypertension involves an impairment of renal sodium excretion along with activation of pro\u2010inflammatory responses in the kidney. Dendritic cells (DCs) are professional antigen\u2010presenting cells that sense disturbances in cell and tissue homeostasis to orchestrate T cell activation and recruitment. Renal DCs distribute as a cellular network throughout the renal interstitium, forming an interface between innate and adaptive immunity. Here, we explore what is known about renal cross\u2010talk following prohypertensive stimuli, which modify renal DCs, T cell polarization, and renal function, leading to hypertension. We also present the phenotypic and functional heterogeneity of DCs, including literature on in\u00a0vitro and in\u00a0vivo signaling mechanisms in a variety of hypertensive models. Current studies indicate that renal sodium retention caused by prohypertensive stimuli requires DC function. The prohypertensive action of DCs also requires T cell function. Several cross\u2010talk mechanisms have been proposed, such as signals from the kidney to the DC, including salt activation of DC's via RAAS receptor signaling, and DC\u2010dependent recruitment of T cells to the kidney then promoting increased renal tubular sodium reabsorption. Knowledge gaps include specific functions of renal DC subtypes, sex differences in DC\u2010mediated hypertension, and clinical studies on DC function in hypertension. The identification of mechanisms leading to renal DCs activation in hypertension may offer new strategies for the prevention and treatment of hypertension.<\/jats:p>","DOI":"10.1111\/apha.70308","type":"journal-article","created":{"date-parts":[[2026,10,8]],"date-time":"2026-10-08T05:35:09Z","timestamp":1791437709000},"update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":0,"title":["Renal Myeloid Mechanisms, Cross\u2010Talk in Hypertension"],"prefix":"10.1111","volume":"242","author":[{"ORCID":"https:\/\/orcid.org\/0000-0002-3411-1222","authenticated-orcid":false,"given":"Luis","family":"Michea","sequence":"first","affiliation":[{"name":"Instituto de Ciencias Biom\u00e9dicas and Departamento de Medicina Interna Norte, Facultad de Medicina Universidad de Chile  Santiago Chile"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"ORCID":"https:\/\/orcid.org\/0000-0002-4589-1738","authenticated-orcid":false,"given":"Heddwen 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