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A considerable part of the population carries\n                    <jats:italic>Candida<\/jats:italic>\n                    in the oral cavity, but only few develop OC. Therefore, the pathogenesis of OC may depend on factors other than the attributes of the fungus, such as host factors and other predisposing factors. Mucosal trauma and inflammation compromise epithelial integrity, fostering a conducive environment for fungal invasion. Molecular insights into the immunocompromised state reveal dysregulation in innate and adaptive immunity, creating a permissive environment for\n                    <jats:italic>Candida<\/jats:italic>\n                    proliferation. Detailed examination of\n                    <jats:italic>Candida<\/jats:italic>\n                    species (spp.) and their virulence factors uncovers a nuanced understanding beyond traditional\n                    <jats:italic>C. albicans<\/jats:italic>\n                    focus, which embrace diverse\n                    <jats:italic>Candida<\/jats:italic>\n                    spp. and their strategies, influencing adhesion, invasion, immune evasion, and biofilm formation. Understanding the pathophysiological microenvironments in OC is crucial for the development of targeted therapeutic interventions. This review aims to unravel the diverse pathophysiological microenvironments influencing OC development focusing on microbial, host, and predisposing factors, and considers\n                    <jats:italic>Candida<\/jats:italic>\n                    resistance to antifungal therapy. The comprehensive approach offers a refined perspective on OC, seeking briefly to identify potential therapeutic targets for future effective management.\n                  <\/jats:p>","DOI":"10.1111\/apm.13412","type":"journal-article","created":{"date-parts":[[2024,4,4]],"date-time":"2024-04-04T03:23:25Z","timestamp":1712201005000},"page":"956-973","update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":34,"title":["Pathophysiological microenvironments in oral candidiasis"],"prefix":"10.1111","volume":"132","author":[{"ORCID":"https:\/\/orcid.org\/0009-0006-0417-7198","authenticated-orcid":false,"given":"Mette Rose","family":"J\u00f8rgensen","sequence":"first","affiliation":[{"name":"Section of Oral Pathology and Oral Medicine, Department of Odontology, Faculty of Health and Medical Sciences University of Copenhagen  Copenhagen Denmark"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"311","published-online":{"date-parts":[[2024,4,4]]},"reference":[{"key":"e_1_2_9_2_1","doi-asserted-by":"publisher","DOI":"10.1111\/odi.12546"},{"key":"e_1_2_9_3_1","doi-asserted-by":"publisher","DOI":"10.3390\/jof7070555"},{"key":"e_1_2_9_4_1","doi-asserted-by":"publisher","DOI":"10.3390\/jof6010015"},{"key":"e_1_2_9_5_1","doi-asserted-by":"publisher","DOI":"10.1016\/0030-4220(92)90191-R"},{"key":"e_1_2_9_6_1","doi-asserted-by":"publisher","DOI":"10.1186\/s12903-019-0775-8"},{"key":"e_1_2_9_7_1","doi-asserted-by":"publisher","DOI":"10.1016\/j.mycmed.2015.12.008"},{"key":"e_1_2_9_8_1","doi-asserted-by":"publisher","DOI":"10.1111\/j.1600-0714.2010.00937.x"},{"key":"e_1_2_9_9_1","first-page":"415","article-title":"Superficial oral fungal infections","volume":"1","author":"Samaranayake LP","year":"1991","journal-title":"Curr Opin Dent"},{"key":"e_1_2_9_10_1","unstructured":"Centers for Disease Control and Prevention (U.S.).Antibiotic resistance threats in the United States 2019. 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