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One hundred and thirteen adult ITP patients with AIS were included, and each ITP\u2010AIS was paired with three control subjects. The ITP\u2010AIS cohort had a median age of 65 (24\u201397) years, was predominately female (61 vs. 52) and had a median platelet count of 44\u2009\u00d7\u200910\n                    <jats:sup>9<\/jats:sup>\n                    \/L (range: 4\u2013355\u2009\u00d7\u200910\n                    <jats:sup>9<\/jats:sup>\n                    \/L) at the time of AIS. Post\u2010infarction treatment included anti\u2010platelet therapy (\n                    <jats:italic>n<\/jats:italic>\n                    \u2009=\u200936, 31.9%), anti\u2010coagulation therapy (\n                    <jats:italic>n<\/jats:italic>\n                    \u2009=\u20094, 3.5%), combined anti\u2010platelet\u2013anti\u2010coagulation therapy (\n                    <jats:italic>n<\/jats:italic>\n                    \u2009=\u20094, 3.5%) and endovascular thrombectomy (\n                    <jats:italic>n<\/jats:italic>\n                    \u2009=\u20091, 0.9%). Haemorrhagic transformation occurred in 5 (4.4%) patients. Three patients (2.7%) died due to intracranial haemorrhage, gastrointestinal haemorrhage or severe pulmonary infection. Multivariate analysis revealed that age greater than 60\u2009years (\n                    <jats:italic>p<\/jats:italic>\n                    \u2009&lt;\u20090.001; odds ratio [OR] 3.12; 95% confidence interval [CI] 1.82\u20135.37), diabetes mellitus (\n                    <jats:italic>p<\/jats:italic>\n                    \u2009=\u20090.018; OR 1.99; 95% CI 1.13\u20133.52), hypertension (\n                    <jats:italic>p<\/jats:italic>\n                    \u2009&lt;\u20090.001; OR 2.75; 95% CI 1.62\u20134.66), renal failure (\n                    <jats:italic>p<\/jats:italic>\n                    \u2009=\u20090.036; OR 3.00; 95% CI 1.08\u20138.38), smoking (\n                    <jats:italic>p<\/jats:italic>\n                    \u2009&lt;\u20090.001; OR 5.02; 95% CI 2.31\u201310.88) and hyper\u2010low\u2010density lipoprotein (LDL)\u2010C (\n                    <jats:italic>p<\/jats:italic>\n                    \u2009=\u20090.02; OR 2.34; 95% CI, 1.14\u20134.78) were independent risk factors for AIS after ITP. This study revealed that ITP\u2010related treatments might not increase the risk of ITP\u2010AIS. The management of ITP\u2010AIS patients requires close collaboration between haematologists and vascular neurologists to carefully balance therapeutic benefits against the risk of haemorrhagic complications.\n                  <\/jats:p>","DOI":"10.1111\/bjh.70572","type":"journal-article","created":{"date-parts":[[2026,6,1]],"date-time":"2026-06-01T10:41:40Z","timestamp":1780310500000},"page":"228-239","update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":0,"title":["Clinical profiling and outcomes of ischaemic stroke patients with immune thrombocytopenia: A multicentre retrospective study"],"prefix":"10.1111","volume":"209","author":[{"given":"LiQin","family":"Cao","sequence":"first","affiliation":[{"name":"Peking University People's Hospital Peking University Institute of Hematology  Beijing China"},{"name":"Collaborative Innovation Center of Hematology Peking University  Beijing China"},{"name":"Beijing Key Laboratory of 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