{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,2,13]],"date-time":"2026-02-13T23:05:24Z","timestamp":1771023924850,"version":"3.50.1"},"reference-count":14,"publisher":"Wiley","issue":"6","license":[{"start":{"date-parts":[[2016,1,13]],"date-time":"2016-01-13T00:00:00Z","timestamp":1452643200000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Clin Physio Funct Imaging"],"published-print":{"date-parts":[[2017,11]]},"abstract":"<jats:title>Summary<\/jats:title><jats:sec><jats:title>Objectives<\/jats:title><jats:p>To evaluate intra\u2010atrial conduction delay in patients with atrial fibrillation (<jats:styled-content style=\"fixed-case\">AF<\/jats:styled-content>) via calculation of conduction velocities (<jats:styled-content style=\"fixed-case\">CV<\/jats:styled-content>s) of the right and left atria.<\/jats:p><\/jats:sec><jats:sec><jats:title>Methods<\/jats:title><jats:p>Electroanatomic mapping was performed during sinus rhythm, in the right atrium (<jats:styled-content style=\"fixed-case\">RA<\/jats:styled-content>) in eight patients with paroxysmal <jats:styled-content style=\"fixed-case\">AF<\/jats:styled-content>, in 12 controls with atrioventricular nodal re\u2010entrant tachycardia (<jats:styled-content style=\"fixed-case\">AVNRT<\/jats:styled-content>) and in the left atrium (<jats:styled-content style=\"fixed-case\">LA<\/jats:styled-content>) in additional 16 <jats:styled-content style=\"fixed-case\">AF<\/jats:styled-content> patients. Three\u2010dimensional maps of activation sequences of the <jats:styled-content style=\"fixed-case\">RA<\/jats:styled-content> and <jats:styled-content style=\"fixed-case\">LA<\/jats:styled-content> were obtained. Local <jats:styled-content style=\"fixed-case\">CV<\/jats:styled-content>s were specifically calculated in the direction of wave\u2010front propagation on the activation maps by using 3\u2010dimensional coordinates and local activation times of triads of sites. Average <jats:styled-content style=\"fixed-case\">CV<\/jats:styled-content>s of each atrium and each of 8 predefined <jats:styled-content style=\"fixed-case\">RA<\/jats:styled-content> and <jats:styled-content style=\"fixed-case\">LA<\/jats:styled-content> regions were calculated.<\/jats:p><\/jats:sec><jats:sec><jats:title>Results<\/jats:title><jats:p>During sinus rhythm, the average <jats:styled-content style=\"fixed-case\">CV<\/jats:styled-content>s of the <jats:styled-content style=\"fixed-case\">RA<\/jats:styled-content> were significantly slower (<jats:italic>P<\/jats:italic>&lt;0\u00b705) in the <jats:styled-content style=\"fixed-case\">AF<\/jats:styled-content> group (0\u00b760\u00a0\u00b1\u00a00\u00b712\u00a0m\u00a0s<jats:sup>\u22121<\/jats:sup>) than in the controls (0\u00b783\u00a0\u00b1\u00a00\u00b713\u00a0m\u00a0s<jats:sup>\u22121<\/jats:sup>). The average <jats:styled-content style=\"fixed-case\">CV<\/jats:styled-content>s of the <jats:styled-content style=\"fixed-case\">RA<\/jats:styled-content> basal, septal and annulus regions were significantly slower than the corresponding regions in controls (<jats:italic>P<\/jats:italic>&lt;0\u00b705). In patients with <jats:styled-content style=\"fixed-case\">AF<\/jats:styled-content>, the average <jats:styled-content style=\"fixed-case\">CV<\/jats:styled-content> of the <jats:styled-content style=\"fixed-case\">LA<\/jats:styled-content> was 0\u00b751\u00a0\u00b1\u00a00\u00b711\u00a0m\u00a0s<jats:sup>\u22121<\/jats:sup>, which is significantly slower than that of the <jats:styled-content style=\"fixed-case\">RA<\/jats:styled-content> and than that of <jats:styled-content style=\"fixed-case\">LA<\/jats:styled-content> as previously reported (<jats:italic>P<\/jats:italic>&lt;0\u00b705).<\/jats:p><\/jats:sec><jats:sec><jats:title>Conclusion<\/jats:title><jats:p>Compared to patients with <jats:styled-content style=\"fixed-case\">AVNRT<\/jats:styled-content>, patients with <jats:styled-content style=\"fixed-case\">AF<\/jats:styled-content> are associated with conduction delay in both atria with the delay being more marked in the <jats:styled-content style=\"fixed-case\">LA<\/jats:styled-content> than in the <jats:styled-content style=\"fixed-case\">RA<\/jats:styled-content>, which suggests the involvement of conduction disturbances in the genesis and\/or perpetuation of <jats:styled-content style=\"fixed-case\">AF<\/jats:styled-content>.<\/jats:p><\/jats:sec>","DOI":"10.1111\/cpf.12342","type":"journal-article","created":{"date-parts":[[2016,1,14]],"date-time":"2016-01-14T03:31:21Z","timestamp":1452742281000},"page":"596-601","source":"Crossref","is-referenced-by-count":39,"title":["Atrial average conduction velocity in patients with and without paroxysmal atrial fibrillation"],"prefix":"10.1111","volume":"37","author":[{"given":"Yaxi","family":"Zheng","sequence":"first","affiliation":[{"name":"Section\u00a0of Arrhythmias Sk\u00e5ne University Hospital Lund University Lund Sweden"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Yunlong","family":"Xia","sequence":"additional","affiliation":[{"name":"Section\u00a0of 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