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With the deepening of research, the pathophysiological mechanisms of endogenous H\n                    <jats:sub>2<\/jats:sub>\n                    S in cancer have been increasingly elucidated: (1) promote angiogenesis, (2) stimulate cell bioenergetics, (3) promote migration and proliferation thereby invasion, (4) inhibit apoptosis and (5) activate abnormal cell cycle. However, the increasing H\n                    <jats:sub>2<\/jats:sub>\n                    S levels via exogenous sources show the opposite trend. This phenomenon can be explained by the bell\u2010shaped pharmacological model of H\n                    <jats:sub>2<\/jats:sub>\n                    S, that is, the production of endogenous (low concentration) H\n                    <jats:sub>2<\/jats:sub>\n                    S promotes tumour growth while the exogenous (high concentration) H\n                    <jats:sub>2<\/jats:sub>\n                    S inhibits tumour growth. Here, we review the impact of endogenous H\n                    <jats:sub>2<\/jats:sub>\n                    S synthesis and metabolism on tumour progression, summarize the mechanism of action of H\n                    <jats:sub>2<\/jats:sub>\n                    S in tumour growth, and discuss the possibility of H\n                    <jats:sub>2<\/jats:sub>\n                    S as a potential target for tumour treatment.\n                  <\/jats:p>","DOI":"10.1111\/cpr.13449","type":"journal-article","created":{"date-parts":[[2023,3,17]],"date-time":"2023-03-17T00:09:11Z","timestamp":1679011751000},"update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":36,"title":["Recent advances in the role of endogenous hydrogen sulphide in cancer 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