{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2025,5,14]],"date-time":"2025-05-14T01:18:23Z","timestamp":1747185503344,"version":"3.40.5"},"reference-count":32,"publisher":"Wiley","issue":"9","license":[{"start":{"date-parts":[[2022,8,25]],"date-time":"2022-08-25T00:00:00Z","timestamp":1661385600000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/creativecommons.org\/licenses\/by-nc-nd\/4.0\/"}],"content-domain":{"domain":["onlinelibrary.wiley.com"],"crossmark-restriction":true},"short-container-title":["Internal Medicine Journal"],"published-print":{"date-parts":[[2023,9]]},"abstract":"<jats:title>Abstract<\/jats:title><jats:sec><jats:title>Background<\/jats:title><jats:p>Ventricular arrhythmia (VA) is the most common cause of sudden cardiac death post\u2010ST elevation myocardial infarction (STEMI). Ventricular tachycardia (VT) may be inducible in electrophysiology studies (EPS) early (&lt;40\u2009days) post\u2010STEMI. Whether it originates from the infarct site remains unknown. We examined the correlation between inducible VT and infarct location post\u2010STEMI.<\/jats:p><\/jats:sec><jats:sec><jats:title>Aims<\/jats:title><jats:p>To investigate the correlation between inducible VT and infarct location post\u2010STEMI.<\/jats:p><\/jats:sec><jats:sec><jats:title>Methods<\/jats:title><jats:p>We retrospectively analysed 46 patients from 2005 to 2017 with STEMI who underwent early programmed ventricular stimulation through EPS (&gt;48 h post\u2010STEMI and &lt;40\u2009days from admission). Gated heart pool scans were used to visualise infarct scar regions, and VT exit sites were derived from induction 12\u2010lead electrocardiography. Patients were followed up for primary outcomes of recurrent VA and all\u2010cause mortality.<\/jats:p><\/jats:sec><jats:sec><jats:title>Results<\/jats:title><jats:p>Forty\u2010six patients were included for analysis, with 50 uniquely induced VT exit sites. Mean left ventricular ejection fraction was 30 \u00b1\u20098.7% and 22% had impaired right ventricular ejection fraction. Mean time from presentation to EPS was 16 \u00b1\u200931.3\u2009days. Of the induced VT, 44 (88%) were from within scar and scar\u2010border regions, whereas 6 (12%) of the induced VT were found to be remote to imaging\u2010derived scar. Over a median follow\u2010up period of 75 months, 6 (13%) patients died, and 7 (15%) patients had recurrent VA. No deaths occurred in patients with remote VT.<\/jats:p><\/jats:sec><jats:sec><jats:title>Conclusion<\/jats:title><jats:p>The majority of early inducible post\u2010infarct VT arises from acute myocardial scar; however, a small portion arises from sites remote from scars with a possible focal aetiology.<\/jats:p><\/jats:sec>","DOI":"10.1111\/imj.15891","type":"journal-article","created":{"date-parts":[[2022,8,1]],"date-time":"2022-08-01T04:39:58Z","timestamp":1659328798000},"page":"1570-1580","update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":0,"title":["Correlation of exit sites of inducible ventricular tachycardia post\u2010<scp>ST<\/scp> elevation myocardial infarction on electrophysiology study, with region of infarct"],"prefix":"10.1111","volume":"53","author":[{"ORCID":"https:\/\/orcid.org\/0000-0003-4689-9982","authenticated-orcid":false,"given":"Karan","family":"Rao","sequence":"first","affiliation":[{"name":"Department of 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