{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2025,6,26]],"date-time":"2025-06-26T04:09:01Z","timestamp":1750910941967,"version":"3.41.0"},"reference-count":14,"publisher":"Wiley","issue":"4","license":[{"start":{"date-parts":[[2017,9,15]],"date-time":"2017-09-15T00:00:00Z","timestamp":1505433600000},"content-version":"vor","delay-in-days":17850,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Australasian Annals of Medicine"],"published-print":{"date-parts":[[1968,11]]},"abstract":"<jats:title>SUMMARY<\/jats:title><jats:p>A case of traumatic renal artery thrombosis in which the patient developed the hyponatnemic hypertensive syndrome is described. Decreased vascular sensitivity to angiotensin, grossly elevated plasma renin levels, hypertension and hyponatr\u00e6mia returned to normal after unilateral nephrectomy. Sequential renin and electrolyte studies indicated that this syndrome was caused by the pressor and natriuretic actions of high circulating levels of renin and angiotensin. No evidence could be found to support the suggestion that the high renin levels were secondary to sodium depletion. A positive result to a phentolamine test was recorded, leading to initial problems in diagnosis. The patient is normotensive and well three years after this episode.<\/jats:p>","DOI":"10.1111\/imj.1968.17.4.315","type":"journal-article","created":{"date-parts":[[2017,9,15]],"date-time":"2017-09-15T08:50:39Z","timestamp":1505465439000},"page":"315-319","source":"Crossref","is-referenced-by-count":3,"title":["HYPONATR\u00c6MIA AND RENOVASCULAR HYPERTENSION"],"prefix":"10.1111","volume":"17","author":[{"given":"J. MARK","family":"BONNIN","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"R. G.","family":"EDWARDS","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"G. C.","family":"SCROOP","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"S. L.","family":"SKINNER","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"R. F.","family":"WHELAN","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"311","published-online":{"date-parts":[[2017,9,15]]},"reference":[{"key":"e_1_2_1_2_1","doi-asserted-by":"publisher","DOI":"10.1016\/0002-9343(66)90107-0"},{"key":"e_1_2_1_3_1","doi-asserted-by":"publisher","DOI":"10.1136\/bmj.2.5454.144"},{"key":"e_1_2_1_4_1","doi-asserted-by":"publisher","DOI":"10.1136\/pgmj.42.485.153"},{"key":"e_1_2_1_5_1","doi-asserted-by":"publisher","DOI":"10.1113\/jphysiol.1964.sp007364"},{"volume-title":"Studies on Some Cardiovascular Drugs in Man","year":"1964","author":"HODGE R. L.","key":"e_1_2_1_6_1"},{"key":"e_1_2_1_7_1","doi-asserted-by":"publisher","DOI":"10.1136\/bmj.1.5531.18"},{"key":"e_1_2_1_8_1","doi-asserted-by":"crossref","first-page":"918","DOI":"10.1001\/jama.1967.03130120026007","article-title":"Oral Contraceptives: Renin Aldosterone and High Blood Pressure","volume":"201","author":"LARAGH J. 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