{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2023,10,16]],"date-time":"2023-10-16T05:15:04Z","timestamp":1697433304245},"reference-count":41,"publisher":"Wiley","issue":"4","license":[{"start":{"date-parts":[[2004,7,29]],"date-time":"2004-07-29T00:00:00Z","timestamp":1091059200000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Acta Physiologica Scandinavica"],"published-print":{"date-parts":[[2004,8]]},"abstract":"<jats:title>Abstract<\/jats:title><jats:p>Hyperprostaglandin E syndrome\/antenatal Bartter syndrome is characterized by NaCl wasting and volume depletion, juxtaglomerula hypertrophy, hyper\u2010reninism and secondary hyperaldosteronism. Primary causes are mutations in the gene for Na\u2010K\u20102Cl\u2010cotransporter, NKCC2, or for potassium channel, ROMK, responsible for medullary NaCl malabsorption. Most intriguing aspect of the syndrome is the association with a massively increased renal prostaglandin production which contributes substantially to the clinical picture of the patients. Therefore the term hyperprostaglandin E syndrome has been introduced. It is unclear how prostaglandins aggravate the NaCl transport deficiency. Aspects to prostaglandin synthesis and receptor\u2010mediated function within the kidney in patients suffering from hyperprostaglandin E syndrome\/antenatal Bartter syndrome will be discussed.<\/jats:p>","DOI":"10.1111\/j.1365-201x.2004.01326.x","type":"journal-article","created":{"date-parts":[[2004,7,30]],"date-time":"2004-07-30T05:29:43Z","timestamp":1091165383000},"page":"523-528","source":"Crossref","is-referenced-by-count":8,"title":["The role of cyclooxygenases and prostanoid receptorsin furosemide\u2010like salt losing tubulopathy:the hyperprostaglandin E syndrome"],"prefix":"10.1111","volume":"181","author":[{"given":"R. M.","family":"N\u00fcsing","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"H. 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