{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2024,5,12]],"date-time":"2024-05-12T18:54:14Z","timestamp":1715540054838},"reference-count":30,"publisher":"Wiley","issue":"6","license":[{"start":{"date-parts":[[2008,3,17]],"date-time":"2008-03-17T00:00:00Z","timestamp":1205712000000},"content-version":"vor","delay-in-days":9603,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Clinical Endocrinology"],"published-print":{"date-parts":[[1981,12]]},"abstract":"<jats:title>SUMMARY<\/jats:title><jats:p>To study effects on pituitary\u2010adrenocortical activity of a sustained block of angiotensin II formation, six \u2018drug\u2010resistant\u2019 patients with essential hypertension were studied before and during treatment with an inhibitor of the angiotensin\u2010converting enzyme (Captopril, SQ 14,225). The drug was given in increasing doses (100\u2013400 mg\/day) for 2 weeks whilst patients received a moderately restricted sodium intake (60\u201380 mmol\/day). Immunoreactive ACTH, cortisol, aldosterone, plasma renin activity (PRA) and the activity of the angiotensin\u2010converting enzyme (ACE) were measured in blood samples drawn at 0800\u20130900 h. Urinary excretion of cortisol and aldosterone were measured in 24\u2010h urine collections. Further information on pituitary\u2010adrenocortical function was obtained by measuring serial plasma corticosteroid levels after submaximal stimulation with a synthetic ACTH preparation. ACTH and cortisol did not change an observation which does not support the hypothesis that glucocorticoid activity is influenced by a decrease in plasma angiotensin II concentrations.<\/jats:p>","DOI":"10.1111\/j.1365-2265.1981.tb00701.x","type":"journal-article","created":{"date-parts":[[2008,3,17]],"date-time":"2008-03-17T13:04:03Z","timestamp":1205759043000},"page":"555-565","source":"Crossref","is-referenced-by-count":1,"title":["PITUITARY\u2014ADRENOCORTICAL FUNCTION IN PATIENTS DURING TREATMENT WITH THE ANGIOTENSIN\u2010CONVERTING ENZYME INHIBITOR CAPTOPRIL"],"prefix":"10.1111","volume":"15","author":[{"given":"A.","family":"ANGELI","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"D.","family":"BISBOCCI","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"F.","family":"ORLANDI","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"P.","family":"PACCOTTI","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"R.","family":"TABASSO","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"C.","family":"TAMAGNONE","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"G. 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