{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,9,1]],"date-time":"2026-09-01T23:50:12Z","timestamp":1788306612023,"version":"build-2803163510"},"reference-count":19,"publisher":"Wiley","issue":"10","license":[{"start":{"date-parts":[[2007,10,1]],"date-time":"2007-10-01T00:00:00Z","timestamp":1191196800000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":["onlinelibrary.wiley.com"],"crossmark-restriction":true},"short-container-title":["Int J Dermatology"],"published-print":{"date-parts":[[2007,10]]},"abstract":"<jats:title>Abstract<\/jats:title><jats:p><jats:bold>Background\u2003<\/jats:bold> Aplasia cutis congenita (ACC) is characterized by the absence of a portion of skin in a localized or widespread area at birth. It manifests usually as a solitary defect on the scalp, but sometimes may occur on the face, trunk, or limbs. ACC is most often a benign isolated defect, but can be associated with other physical anomalies or malformation syndromes. A few cases have been reported in which patients with split cord malformation (SCM) have presented with ACC.<\/jats:p><jats:p><jats:bold>Methods\u2003<\/jats:bold> Two patients with SCM are reported.<\/jats:p><jats:p><jats:bold>Results\u2003<\/jats:bold> Both patients presented with ACC and abnormal hair growth on their backs. Type II SCM was detected in the first patient and Type I SCM in the second. No surgical treatment was performed because the patients were neurologically intact.<\/jats:p><jats:p><jats:bold>Conclusions\u2003<\/jats:bold> ACC may seldom manifest as a skin marker of SCM. Our patients are unique examples of SCM presenting with ACC, but not requiring surgery. It is important to recognize ACC as a cutaneous sign of SCM, and to refer these patients to radiologic evaluation as soon as possible.<\/jats:p>","DOI":"10.1111\/j.1365-4632.2007.03212.x","type":"journal-article","created":{"date-parts":[[2007,10,3]],"date-time":"2007-10-03T07:00:55Z","timestamp":1191394855000},"page":"1031-1035","update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":12,"title":["Aplasia cutis congenita: a rare cutaneous sign of split cord malformations"],"prefix":"10.1111","volume":"46","author":[{"given":"Yusuf","family":"Izci","sequence":"first","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"M\u00fczeyyen","family":"G\u00f6n\u00fcl","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Halil Ibrahim","family":"Secer","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Engin","family":"G\u00f6n\u00fcl","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"311","published-online":{"date-parts":[[2007,10]]},"reference":[{"key":"e_1_2_4_2_2","doi-asserted-by":"publisher","DOI":"10.1227\/00006123-199209000-00010"},{"key":"e_1_2_4_3_2","doi-asserted-by":"publisher","DOI":"10.1227\/00006123-199209000-00011"},{"key":"e_1_2_4_4_2","doi-asserted-by":"publisher","DOI":"10.1007\/s10143-003-0313-6"},{"key":"e_1_2_4_5_2","doi-asserted-by":"publisher","DOI":"10.3171\/jns.1998.88.1.0057"},{"key":"e_1_2_4_6_2","doi-asserted-by":"publisher","DOI":"10.1159\/000080859"},{"key":"e_1_2_4_7_2","doi-asserted-by":"publisher","DOI":"10.1016\/S0190-9622(86)70082-0"},{"key":"e_1_2_4_8_2","doi-asserted-by":"publisher","DOI":"10.1111\/j.0736-8046.2004.21213.x"},{"key":"e_1_2_4_9_2","unstructured":"OllivierCP.Trait\u00e9 des maladies de la moelle \u00e9pini\u00e8re. 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