{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,11,6]],"date-time":"2026-11-06T13:28:23Z","timestamp":1793971703374,"version":"4.8.0"},"reference-count":33,"publisher":"Wiley","issue":"2","license":[{"start":{"date-parts":[[2006,12,4]],"date-time":"2006-12-04T00:00:00Z","timestamp":1165190400000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":["obgyn.onlinelibrary.wiley.com"],"crossmark-restriction":true},"short-container-title":["BJOG"],"published-print":{"date-parts":[[2007,2]]},"abstract":"<jats:p>\n                    <jats:bold>Objective\u2002<\/jats:bold>\n                    To evaluate the risk of preterm delivery in patients with adenomyosis.\n                  <\/jats:p>\n                  <jats:p>\n                    <jats:bold>Design\u2002<\/jats:bold>\n                    A 1:2 nested case\u2013control study.\n                  <\/jats:p>\n                  <jats:p>\n                    <jats:bold>Setting\u2002<\/jats:bold>\n                    Tertiary\u2010care institution.\n                  <\/jats:p>\n                  <jats:p>\n                    <jats:bold>Population\u2002<\/jats:bold>\n                    A base cohort population of 2138 pregnant women who attended routine prenatal check\u2010up between July 1999 and June 2005.\n                  <\/jats:p>\n                  <jats:p>\n                    <jats:bold>Methods\u2002<\/jats:bold>\n                    From this base cohort population, gravid women with singleton pregnancy who delivered prior to the completion of 37 weeks of gestation were identified and formed the study group. Singleton gravid women who had term delivery and who matched with age, body mass index, smoking, and status of previous preterm delivery were recruited concurrently and served as control group. Preterm delivery cases were further divided into spontaneous preterm delivery and preterm premature rupture of membranes (PPROM) cases.\n                  <\/jats:p>\n                  <jats:p>\n                    <jats:bold>Main outcome measures\u2002<\/jats:bold>\n                    Risk analysis of preterm delivery between gravid women with and without adenomyosis.\n                  <\/jats:p>\n                  <jats:p>\n                    <jats:bold>Results\u2002<\/jats:bold>\n                    One\u2010hundred and four preterm delivery case subjects and 208 control subjects were assessed. Overall, gravid women with adenomyosis were associated with significantly increased risk of preterm delivery (adjusted odds ratio 1.96, 95% CI 1.23\u20134.47,\n                    <jats:italic>P<\/jats:italic>\n                    = 0.022). For subgroup analysis, gravid women with adenomyosis had an adjusted 1.84\u2010fold risk of spontaneous preterm delivery (95% CI 1.32\u20134.31,\n                    <jats:italic>P<\/jats:italic>\n                    = 0.012) and an adjusted 1.98\u2010fold risk of PPROM (95% CI 1.39\u20133.15,\n                    <jats:italic>P<\/jats:italic>\n                    = 0.017).\n                  <\/jats:p>\n                  <jats:p>\n                    <jats:bold>Conclusions\u2002<\/jats:bold>\n                    Gravid women with adenomyosis were associated with increased risk of both spontaneous preterm delivery and PPROM. A common pathophysiological pathway may exist in these two disorders. Further in\u2010depth biochemical and molecular studies are necessary to explore this phenomenon.\n                  <\/jats:p>","DOI":"10.1111\/j.1471-0528.2006.01186.x","type":"journal-article","created":{"date-parts":[[2006,12,5]],"date-time":"2006-12-05T11:04:39Z","timestamp":1165316679000},"page":"165-169","update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":139,"title":["Adenomyosis and risk of preterm delivery"],"prefix":"10.1111","volume":"114","author":[{"given":"C\u2010M","family":"Juang","sequence":"first","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"P","family":"Chou","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"M\u2010S","family":"Yen","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"N\u2010F","family":"Twu","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"H\u2010C","family":"Horng","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"W\u2010L","family":"Hsu","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"311","published-online":{"date-parts":[[2007,1,9]]},"reference":[{"key":"e_1_2_6_2_2","first-page":"424","article-title":"Adenomyosis: a neglected diagnosis","volume":"50","author":"Owolabi TO","year":"1977","journal-title":"Obstet Gynecol"},{"key":"e_1_2_6_3_2","doi-asserted-by":"publisher","DOI":"10.1016\/S0029-7844(99)00659-6"},{"key":"e_1_2_6_4_2","doi-asserted-by":"publisher","DOI":"10.1093\/humrep\/12.6.1275"},{"key":"e_1_2_6_5_2","doi-asserted-by":"publisher","DOI":"10.1067\/mob.2002.124285"},{"key":"e_1_2_6_6_2","doi-asserted-by":"publisher","DOI":"10.1056\/NEJM198501103120204"},{"key":"e_1_2_6_7_2","first-page":"262","article-title":"Infection in the pathogenesis of preterm labor","volume":"12","author":"Romero R","year":"1988","journal-title":"Semin Perinatol"},{"key":"e_1_2_6_8_2","doi-asserted-by":"publisher","DOI":"10.1093\/oxfordjournals.epirev.a036128"},{"issue":"5","key":"e_1_2_6_9_2","first-page":"750","article-title":"Clinical and environmental predictors of preterm labor","volume":"81","author":"Heffner LJ","year":"1993","journal-title":"Obstet Gynecol"},{"key":"e_1_2_6_10_2","unstructured":"American College of Obstetricians and Gynecologists.Preterm labor. 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