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B\u2010cell malignancies are particularly affected by oncogenic mutations, translocations, and copy number alterations affecting key components the NF\u2010\u03baB pathway, most likely owing to the pervasive role of this pathway in normal B cells. These genetic aberrations cause tumors to be \u2018addicted\u2019 to NF\u2010\u03baB, which can be exploited therapeutically. Since each subtype of lymphoid cancer utilizes different mechanisms to activate NF\u2010\u03baB, several different therapeutic strategies are needed to address this pathogenetic heterogeneity. 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