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(2) Do desmogleins adequately address PV pathophysiology? (3) How does acantholysis occur in PV? (4) Is PV still a lethal disease? (5) What is the role of rituximab (RTX) in PV treatment? Results from extensive literature searches suggested the following: (1) Target antigens of PV include a variety of molecules and receptors that are not physically compartmentalized within the epidermis. (2) PV is caused by a variety of autoantibodies to keratinocyte self\u2010antigens, which concur to cause blistering by acting synergistically. (3) The concept of apoptolysis distinguishes the unique mechanism of autoantibody\u2010induced keratinocyte damage in PV from other known forms of cell death. (4) PV remains potentially life\u2010threatening largely because of treatment side effects, but it is uncertain which therapies carry the highest likelihood of lethal risk. (5) RTX is a very promising treatment option in patients with widespread recalcitrant or life\u2010threatening PV. RTX\u2019s cost is an issue, its long\u2010term side effects are still unknown, and randomized controlled trials are needed to establish the optimal dosing regimen.<\/jats:p>","DOI":"10.1111\/j.1601-0825.2011.01899.x","type":"journal-article","created":{"date-parts":[[2011,12,25]],"date-time":"2011-12-25T00:14:35Z","timestamp":1324772075000},"page":"442-458","source":"Crossref","is-referenced-by-count":39,"title":["Urban legends: pemphigus 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