{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,10,19]],"date-time":"2026-10-19T20:42:04Z","timestamp":1792442524546,"version":"4.5.0"},"reference-count":42,"publisher":"Wiley","issue":"7","license":[{"start":{"date-parts":[[2001,7,1]],"date-time":"2001-07-01T00:00:00Z","timestamp":993945600000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["J of Small Animal Practice"],"published-print":{"date-parts":[[2001,7]]},"abstract":"<jats:p>A nine\u2010month\u2010old golden retriever bitch was presented with exercise intolerance and recurrent nasal discharge. Based on clinical, radiographic and ultrasonographic examination, a diagnosis of rhinitis, situs inversus totalis and tricuspid valve insufficiency was established. The results of video\u2010 and electron microscopy studies of the respiratory epithelium were compatible with primary ciliary dyskinesia (PCD). However, no evidence of a primary ultrastructural defect of the cilia was found. The dog was euthanased because of the poor prognosis. At necropsy, a hydrocephalus internus and a subaortic stenosis were additionally diagnosed. PCD, in combination with situs inversus, has been previously reported in golden retrievers, but without a concomitant hydrocephalus internus. Furthermore, concomitant occurrence of internal cardiac malformation and PCD has not previously been reported in the dog.<\/jats:p>","DOI":"10.1111\/j.1748-5827.2001.tb02471.x","type":"journal-article","created":{"date-parts":[[2008,4,10]],"date-time":"2008-04-10T10:12:07Z","timestamp":1207822327000},"page":"345-348","source":"Crossref","is-referenced-by-count":28,"title":["Primary ciliary dyskinesia with situs inversus totalis, hydrocephalus internus and cardiac malformations in a dog"],"prefix":"10.1111","volume":"42","author":[{"given":"I. 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