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There have been many attempts to find a human leukocyte antigen (<jats:styled-content style=\"fixed-case\">HLA<\/jats:styled-content>) association with <jats:styled-content style=\"fixed-case\">GBS<\/jats:styled-content> and <jats:styled-content style=\"fixed-case\">CIDP<\/jats:styled-content> with little success. There have been studies of other plausible genes in <jats:styled-content style=\"fixed-case\">GBS<\/jats:styled-content> and <jats:styled-content style=\"fixed-case\">CIDP<\/jats:styled-content> and the role of these genes in <jats:styled-content style=\"fixed-case\">GBS<\/jats:styled-content> and <jats:styled-content style=\"fixed-case\">CIDP<\/jats:styled-content> and the data from these genetic studies is reviewed. Some of the genes that have been studied are immune related and some others have nervous system effects. The studies are limited by small numbers. Some of the genes show association with disease severity rather than disease susceptibility. The need for more detailed molecular studies of the role of <jats:styled-content style=\"fixed-case\">HLA<\/jats:styled-content> molecules and the need for modern genetic approaches to <jats:styled-content style=\"fixed-case\">GBS<\/jats:styled-content> and <jats:styled-content style=\"fixed-case\">CIDP<\/jats:styled-content> are explained.<\/jats:p>","DOI":"10.1111\/jns5.12074","type":"journal-article","created":{"date-parts":[[2014,7,7]],"date-time":"2014-07-07T17:51:18Z","timestamp":1404755478000},"page":"88-103","source":"Crossref","is-referenced-by-count":65,"title":["Genetics of Guillain\u2010Barr\u00e9 syndrome (<scp>GBS<\/scp>) and chronic inflammatory demyelinating polyradiculoneuropathy (<scp>CIDP<\/scp>): current knowledge and future directions"],"prefix":"10.1111","volume":"19","author":[{"given":"Stefan","family":"Blum","sequence":"first","affiliation":[{"name":"The University of Queensland Centre for Clinical Research UQ CCR  Queensland 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