{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,1,13]],"date-time":"2026-01-13T08:26:22Z","timestamp":1768292782986,"version":"3.49.0"},"reference-count":17,"publisher":"Wiley","issue":"9","license":[{"start":{"date-parts":[[2021,2,2]],"date-time":"2021-02-02T00:00:00Z","timestamp":1612224000000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":["onlinelibrary.wiley.com"],"crossmark-restriction":true},"short-container-title":["Journal of Cardiac Surgery"],"published-print":{"date-parts":[[2022,9]]},"abstract":"<jats:title>Abstract<\/jats:title>\n                  <jats:sec>\n                    <jats:title>Background<\/jats:title>\n                    <jats:p>As transcatheter aortic valve replacement (TAVR) procedures increase, more data is available on the development of conduction abnormalities requiring permanent pacemaker (PPM) implantation post\u2010TAVR. Mechanistically, new pacemaker implantation and incidence of associated tricuspid regurgitation (TR) post\u2010TAVR is not well understood. Studies have evaluated the predictability of patient anatomy towards\u00a0risk for needing permanent pacemaker (PPM) post\u2010TAVR; however, little has been reported on new PPM and TR in patients post\u2010TAVR.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Methods<\/jats:title>\n                    <jats:p>This retrospective study identified patients at our health system who underwent PPM following TAVR from January 2014 to June 2018. Data from both TAVR and PPM procedures as well as patient demographics were collected. Echocardiographic data before TAVR, between TAVR and PPM placement, and the most recent echocardiogram at the time of chart review were analyzed.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Results<\/jats:title>\n                    <jats:p>Of 796 patients who underwent TAVR between January 2014 and June 2018, 89 patients (11%) subsequently required PPM. Out of the 89 patients who required PPM implantation, 82 patients had pre\u2010TAVR and 2\u2010year post\u2010TAVR echocardiographic imaging data. At baseline, 22% (18\/82) of patients had at least moderate TR. At 2\u2010year post\u2010TAVR echocardiographic imaging follow\u2010up; 27% (22\/82) of patients had at least moderate TR. Subgroup analysis was performed according to the TAVR valve size implanted. In patients who received a TAVR device\u2009&lt;\u200929\u2009mm in diameter in size, 25% (11\/44) had worsening TR. In patients who received a TAVR device\u2009\u2265\u200929\u2009mm in diameter, 37% (14\/38) had worsening TR.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Conclusion<\/jats:title>\n                    <jats:p>We have demonstrated a patient population that may be predisposed to developing worsening TR and right heart function after TAVR and Pacemaker implantation.<\/jats:p>\n                  <\/jats:sec>","DOI":"10.1111\/jocs.15363","type":"journal-article","created":{"date-parts":[[2021,2,6]],"date-time":"2021-02-06T04:02:49Z","timestamp":1612584169000},"page":"2937-2942","update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":5,"title":["Pacemaker following transcatheter aortic valve replacement and tricuspid regurgitation:\u00a0A single\u2010center experience"],"prefix":"10.1155","volume":"37","author":[{"ORCID":"https:\/\/orcid.org\/0000-0002-4758-7211","authenticated-orcid":false,"given":"Paul","family":"Nona","sequence":"first","affiliation":[{"name":"Division of Cardiology, Henry Ford Health System Henry Ford Hospital Detroit Michigan USA"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Nicholas","family":"Coriasso","sequence":"additional","affiliation":[{"name":"Division of Cardiology, Henry Ford Health System Henry Ford Hospital Detroit Michigan USA"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Arfaat","family":"Khan","sequence":"additional","affiliation":[{"name":"Division of Cardiology, Henry Ford Health System Henry Ford Hospital Detroit Michigan USA"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Gurjit","family":"Singh","sequence":"additional","affiliation":[{"name":"Division of Cardiology, Henry Ford Health System Henry Ford Hospital Detroit Michigan USA"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Marvin H.","family":"Eng","sequence":"additional","affiliation":[{"name":"Division of Cardiology, Henry Ford Health System Henry Ford Hospital Detroit Michigan USA"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Tiberio","family":"Frisoli","sequence":"additional","affiliation":[{"name":"Division of Cardiology, Henry Ford Health System Henry Ford Hospital Detroit Michigan USA"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Brian P.","family":"O'Neill","sequence":"additional","affiliation":[{"name":"Division of Cardiology, Henry Ford Health System Henry Ford Hospital Detroit Michigan USA"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Pedro A.","family":"Villablanca","sequence":"additional","affiliation":[{"name":"Division of Cardiology, Henry Ford Health System Henry Ford Hospital Detroit Michigan USA"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"James C.","family":"Lee","sequence":"additional","affiliation":[{"name":"Division of Cardiology, Henry Ford Health System Henry Ford Hospital Detroit Michigan USA"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Gordon","family":"Jacobsen","sequence":"additional","affiliation":[{"name":"Department of Public Health Sciences, Henry Ford Health System Henry Ford Hospital Detroit Michigan USA"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"William W.","family":"O'Neill","sequence":"additional","affiliation":[{"name":"Division of Cardiology, Henry Ford Health System Henry Ford Hospital Detroit Michigan USA"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"ORCID":"https:\/\/orcid.org\/0000-0002-5784-9924","authenticated-orcid":false,"given":"Dee Dee","family":"Wang","sequence":"additional","affiliation":[{"name":"Division of Cardiology, Henry Ford Health System Henry Ford Hospital Detroit Michigan USA"}],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"311","published-online":{"date-parts":[[2021,2,2]]},"reference":[{"key":"e_1_2_8_2_1","doi-asserted-by":"publisher","DOI":"10.1002\/ccd.28512"},{"key":"e_1_2_8_3_1","doi-asserted-by":"crossref","DOI":"10.1161\/JAHA.120.018405","article-title":"Myocardial injury after balloon predilatation versus direct transcatheter aortic valve replacement: insights from the DIRECTAVI trial","author":"Akodad M","year":"2020","journal-title":"J Am Heart Assoc"},{"key":"e_1_2_8_4_1","doi-asserted-by":"publisher","DOI":"10.1016\/j.ijcha.2020.100654"},{"key":"e_1_2_8_5_1","unstructured":"HamdanA NassarM SchwammenthalE et al.Short membranous septum length in bicuspid aortic valve stenosis increases the risk of conduction disturbances [published online ahead of print October 29 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