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Members of the<jats:styled-content style=\"fixed-case\">K<jats:sub>V<\/jats:sub><\/jats:styled-content>channel family: (i) are highly expressed in<jats:styled-content style=\"fixed-case\">VSMC<\/jats:styled-content>s; (ii) are active at the resting membrane potential of<jats:styled-content style=\"fixed-case\">VSMC<\/jats:styled-content>s in vivo (\u221245 to \u221230<jats:styled-content style=\"fixed-case\">mV<\/jats:styled-content>); (iii) contribute to the negative feedback regulation of<jats:styled-content style=\"fixed-case\">VSMC<\/jats:styled-content>membrane potential and myogenic tone; (iv) are activated by<jats:styled-content style=\"fixed-case\">cAMP<\/jats:styled-content>\u2010related vasodilators, hydrogen sulfide and hydrogen peroxide; (v) are inhibited by increases in intracellular Ca<jats:sup>2+<\/jats:sup>and vasoconstrictors that signal through G<jats:sub>q<\/jats:sub>\u2010coupled receptors; (vi) are involved in the proliferative phenotype of<jats:styled-content style=\"fixed-case\">VSMC<\/jats:styled-content>s; and (vii) are modulated by diseases such as hypertension, obesity, the metabolic syndrome and diabetes. 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