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Here, we show that <jats:styled-content style=\"fixed-case\"><jats:italic>C<\/jats:italic><\/jats:styled-content><jats:italic>hlamydia<\/jats:italic> infection in human epithelial cells induces microtubule remodeling, in particular the formation of detyrosinated stable <jats:styled-content style=\"fixed-case\">MTs<\/jats:styled-content>, to recruit <jats:styled-content style=\"fixed-case\">G<\/jats:styled-content>olgi ministacks, but not recycling endosomes, to the inclusion. These stable microtubules show increased resistance to chemically induced depolymerization, and their selective depletion results in reduced bacterial infectivity. <jats:styled-content style=\"fixed-case\">R<\/jats:styled-content>ab6 knockdown reversibly prevented not only <jats:styled-content style=\"fixed-case\">G<\/jats:styled-content>olgi ministack formation but also detyrosinated microtubule association with the inclusion. Our data demonstrate that <jats:styled-content style=\"fixed-case\"><jats:italic>C<\/jats:italic><\/jats:styled-content><jats:italic>hlamydia<\/jats:italic> co\u2010opts the function of stable microtubules to support its development.<\/jats:p>","DOI":"10.1111\/mmi.12829","type":"journal-article","created":{"date-parts":[[2014,10,15]],"date-time":"2014-10-15T07:37:40Z","timestamp":1413358660000},"page":"1285-1297","source":"Crossref","is-referenced-by-count":47,"title":["<scp><i>C<\/i><\/scp><i>hlamydia trachomatis<\/i> remodels stable microtubules to coordinate <scp>G<\/scp>olgi stack recruitment to the chlamydial inclusion surface"],"prefix":"10.1111","volume":"94","author":[{"given":"Munir A.","family":"Al\u2010Zeer","sequence":"first","affiliation":[{"name":"Department of Molecular Biology Max Planck Institute for Infection Biology  Charit\u00e8platz\u20091 10117 Berlin Germany"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Hesham 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