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Our study of the mechanisms implicated in\n                    <jats:styled-content style=\"fixed-case\">H<\/jats:styled-content>\n                    x\u2010\n                    <jats:styled-content style=\"fixed-case\">IR<\/jats:styled-content>\n                    against\n                    <jats:italic>\n                      <jats:styled-content style=\"fixed-case\">P<\/jats:styled-content>\n                      seudomonas syringae\n                    <\/jats:italic>\n                    pv.\n                    <jats:italic>tomato<\/jats:italic>\n                    \u2005\n                    <jats:styled-content style=\"fixed-case\">DC<\/jats:styled-content>\n                    3000 suggests that hexanoic acid (\n                    <jats:styled-content style=\"fixed-case\">H<\/jats:styled-content>\n                    x) treatment counteracts the negative effect of coronatine (\n                    <jats:styled-content style=\"fixed-case\">COR<\/jats:styled-content>\n                    ) and jasmonyl\u2010isoleucine (\n                    <jats:styled-content style=\"fixed-case\">JA<\/jats:styled-content>\n                    \u2010\n                    <jats:styled-content style=\"fixed-case\">I<\/jats:styled-content>\n                    le) on the salicylic acid (\n                    <jats:styled-content style=\"fixed-case\">SA<\/jats:styled-content>\n                    ) pathway. In\n                    <jats:styled-content style=\"fixed-case\">H<\/jats:styled-content>\n                    x\u2010treated plants, an increase in the expression of jasmonic acid carboxyl methyltransferase (\n                    <jats:italic>\n                      <jats:styled-content style=\"fixed-case\">JMT<\/jats:styled-content>\n                    <\/jats:italic>\n                    ) and the\n                    <jats:styled-content style=\"fixed-case\">SA<\/jats:styled-content>\n                    marker genes\n                    <jats:italic>\n                      <jats:styled-content style=\"fixed-case\">PR<\/jats:styled-content>\n                      1\n                    <\/jats:italic>\n                    and\n                    <jats:italic>\n                      <jats:styled-content style=\"fixed-case\">PR<\/jats:styled-content>\n                      5\n                    <\/jats:italic>\n                    indicates a boost in this signalling pathway at the expense of a decrease in\n                    <jats:styled-content style=\"fixed-case\">JA<\/jats:styled-content>\n                    \u2010\n                    <jats:styled-content style=\"fixed-case\">I<\/jats:styled-content>\n                    le. Moreover,\n                    <jats:styled-content style=\"fixed-case\">H<\/jats:styled-content>\n                    x treatment potentiates 12\u2010oxo\u2010phytodienoic acid accumulation, which suggests that this molecule might play a role\n                    <jats:italic>per se<\/jats:italic>\n                    in\n                    <jats:styled-content style=\"fixed-case\">H<\/jats:styled-content>\n                    x\u2010\n                    <jats:styled-content style=\"fixed-case\">IR<\/jats:styled-content>\n                    . These results support a positive relationship between the\n                    <jats:styled-content style=\"fixed-case\">SA<\/jats:styled-content>\n                    and\n                    <jats:styled-content style=\"fixed-case\">JA<\/jats:styled-content>\n                    pathways in\n                    <jats:styled-content style=\"fixed-case\">H<\/jats:styled-content>\n                    x\u2010primed plants. Furthermore, one of the mechanisms of virulence mediated by\n                    <jats:styled-content style=\"fixed-case\">COR<\/jats:styled-content>\n                    is stomatal re\u2010opening on infection with\n                    <jats:italic>\n                      <jats:styled-content style=\"fixed-case\">P<\/jats:styled-content>\n                      .\u2009syringae\n                    <\/jats:italic>\n                    . In this work, we observed that\n                    <jats:styled-content style=\"fixed-case\">H<\/jats:styled-content>\n                    x seems to inhibit stomatal opening\n                    <jats:italic>in\u2009planta<\/jats:italic>\n                    in the presence of\n                    <jats:styled-content style=\"fixed-case\">COR<\/jats:styled-content>\n                    , which suggests that, on infection in tomato, this treatment suppresses effector action to prevent bacterial entry into the mesophyll.\n                  <\/jats:p>","DOI":"10.1111\/mpp.12010","type":"journal-article","created":{"date-parts":[[2012,12,24]],"date-time":"2012-12-24T00:11:58Z","timestamp":1356307918000},"page":"342-355","update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":85,"title":["Hexanoic acid is a resistance inducer that protects tomato plants against\n                    <i>\n                      <scp>P<\/scp>\n                      seudomonas syringae\n                    <\/i>\n                    by priming the jasmonic acid and salicylic acid pathways"],"prefix":"10.1111","volume":"14","author":[{"given":"Loredana","family":"Scalschi","sequence":"first","affiliation":[{"name":"Grupo de Bioqu\u00edmica y Biotecnolog\u00eda \u00c1rea de Fisiolog\u00eda Vegetal Departamento de Ciencias Agrarias y del Medio Natural ESTCE Universitat Jaume I  Castell\u00f3n 12071 Spain"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Begonya","family":"Vicedo","sequence":"additional","affiliation":[{"name":"Grupo de Bioqu\u00edmica y Biotecnolog\u00eda \u00c1rea de Fisiolog\u00eda Vegetal Departamento de Ciencias Agrarias y del Medio Natural ESTCE Universitat Jaume I  Castell\u00f3n 12071 Spain"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Gemma","family":"Cama\u00f1es","sequence":"additional","affiliation":[{"name":"Grupo de Bioqu\u00edmica y Biotecnolog\u00eda \u00c1rea de Fisiolog\u00eda 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