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The central nervous system (\n                    <jats:styled-content style=\"fixed-case\">CNS<\/jats:styled-content>\n                    ) has a poor intrinsic capacity for regeneration, although some functional recovery does occur. This is mainly in the form of sprouting, dendritic remodelling and changes in neuronal coding, firing and synaptic properties; elements collectively known as plasticity. An important approach to repair the injured\n                    <jats:styled-content style=\"fixed-case\">CNS<\/jats:styled-content>\n                    is therefore to harness, promote and refine plasticity. In the adult, this is partly limited by the extracellular matrix (\n                    <jats:styled-content style=\"fixed-case\">ECM<\/jats:styled-content>\n                    ). While the\n                    <jats:styled-content style=\"fixed-case\">ECM<\/jats:styled-content>\n                    typically provides a supportive framework to\n                    <jats:styled-content style=\"fixed-case\">CNS<\/jats:styled-content>\n                    neurones, its role is not only structural; the\n                    <jats:styled-content style=\"fixed-case\">ECM<\/jats:styled-content>\n                    is homeostatic, actively regulatory and of great signalling importance, both directly via receptor or coreceptor\u2010mediated action and via spatially and temporally relevant localization of other signalling molecules. In an injury or disease state, the\n                    <jats:styled-content style=\"fixed-case\">ECM<\/jats:styled-content>\n                    represents a key environment to support a healing and\/or regenerative response. However, there are aspects of its composition which prove suboptimal for recovery: some molecules present in the\n                    <jats:styled-content style=\"fixed-case\">ECM<\/jats:styled-content>\n                    restrict plasticity and limit repair. An important therapeutic concept is therefore to render the\n                    <jats:styled-content style=\"fixed-case\">ECM<\/jats:styled-content>\n                    environment more permissive by manipulating key components, such as inhibitory chondroitin sulphate proteoglycans. In this review we discuss the major components of the\n                    <jats:styled-content style=\"fixed-case\">ECM<\/jats:styled-content>\n                    and the role they play during development and following brain or spinal cord injury and we consider a number of experimental strategies which involve manipulations of the\n                    <jats:styled-content style=\"fixed-case\">ECM<\/jats:styled-content>\n                    , with the aim of promoting functional recovery to the injured brain and spinal cord.\n                  <\/jats:p>","DOI":"10.1111\/nan.12114","type":"journal-article","created":{"date-parts":[[2013,12,27]],"date-time":"2013-12-27T05:22:55Z","timestamp":1388121775000},"page":"26-59","source":"Crossref","is-referenced-by-count":151,"title":["Review: Manipulating the extracellular matrix and its role in brain and spinal cord plasticity and repair"],"prefix":"10.1111","volume":"40","author":[{"given":"E. 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