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The 2013\n                    <jats:styled-content style=\"fixed-case\">I<\/jats:styled-content>\n                    nternational\n                    <jats:styled-content style=\"fixed-case\">L<\/jats:styled-content>\n                    eague\n                    <jats:styled-content style=\"fixed-case\">A<\/jats:styled-content>\n                    gainst\n                    <jats:styled-content style=\"fixed-case\">E<\/jats:styled-content>\n                    pilepsy (\n                    <jats:styled-content style=\"fixed-case\">ILAE<\/jats:styled-content>\n                    ) classification segregates\n                    <jats:styled-content style=\"fixed-case\">HS<\/jats:styled-content>\n                    into typical (type 1) and atypical (type 2 and 3) groups, based on the histological patterns of subfield neuronal loss and gliosis. In addition, granule cell reorganization and alterations of interneuronal populations, neuropeptide fibre networks and mossy fibre sprouting are distinctive features of\n                    <jats:styled-content style=\"fixed-case\">HS<\/jats:styled-content>\n                    associated with epilepsies; they can be useful diagnostic aids to discriminate from other causes of\n                    <jats:styled-content style=\"fixed-case\">HS<\/jats:styled-content>\n                    , as well as highlighting potential mechanisms of hippocampal epileptogenesis. The cause of\n                    <jats:styled-content style=\"fixed-case\">HS<\/jats:styled-content>\n                    remains elusive and may be multifactorial; the contribution of febrile seizures, genetic susceptibility, inflammatory and neurodevelopmental factors are discussed.\n                    <jats:italic>Post\u2010mortem<\/jats:italic>\n                    based research in\n                    <jats:styled-content style=\"fixed-case\">HS<\/jats:styled-content>\n                    , as an addition to studies on surgical samples, has the added advantage of enabling the study of the wider network changes associated with\n                    <jats:styled-content style=\"fixed-case\">HS<\/jats:styled-content>\n                    , the long\u2010term effects of epilepsy on the pathology and associated comorbidities. It is likely that\n                    <jats:styled-content style=\"fixed-case\">HS<\/jats:styled-content>\n                    is heterogeneous in aspects of its cause, epileptogenetic mechanisms, network alterations and response to medical and surgical treatments. Future neuropathological studies will contribute to better recognition and understanding of these clinical and patho\u2010aetiological subtypes of\n                    <jats:styled-content style=\"fixed-case\">HS<\/jats:styled-content>\n                    .\n                  <\/jats:p>","DOI":"10.1111\/nan.12150","type":"journal-article","created":{"date-parts":[[2014,4,24]],"date-time":"2014-04-24T23:38:36Z","timestamp":1398382716000},"page":"520-543","source":"Crossref","is-referenced-by-count":531,"title":["Review: Hippocampal sclerosis in epilepsy: a neuropathology review"],"prefix":"10.1111","volume":"40","author":[{"given":"Maria","family":"Thom","sequence":"first","affiliation":[{"name":"Departments of Neuropathology and Clinical and Experimental Epilepsy Institute of Neurology University College London London UK"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"311","published-online":{"date-parts":[[2014,7]]},"reference":[{"key":"e_1_2_9_2_1","first-page":"510","article-title":"De l'\u00e9pilepsie consider\u00e9e dans ses rapports avec l'ali\u00e9nation mentale. 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