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Three neuropathologic subtypes can be recognized, based on the presence of inclusions made of tau isoforms with three and four repeats, predominantly three repeats and mostly four repeats. This is relevant for establishing a correlation between structural magnetic resonance imaging and positron emission tomography using tracers specific for aggregated tau. Longitudinal studies will be essential to determine the evolution of anatomical alterations from the asymptomatic stage to the various phases of disease following the onset of symptoms.\n                  <\/jats:p>","DOI":"10.1111\/nan.12213","type":"journal-article","created":{"date-parts":[[2014,12,31]],"date-time":"2014-12-31T04:14:50Z","timestamp":1419999290000},"page":"24-46","source":"Crossref","is-referenced-by-count":446,"title":["Invited review: Frontotemporal dementia caused by\n                    <i>microtubule\u2010associated protein tau<\/i>\n                    gene (\n                    <scp>\n                      <i>MAPT<\/i>\n                    <\/scp>\n                    ) mutations: a chameleon for neuropathology and neuroimaging"],"prefix":"10.1111","volume":"41","author":[{"given":"Bernardino","family":"Ghetti","sequence":"first","affiliation":[{"name":"Department of Pathology and Laboratory Medicine Indiana University School of Medicine  Indianapolis 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