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Mutations in Elongator genes inhibit\n                          <jats:italic>Xcc<\/jats:italic>\n                          \u2010,\n                          <jats:italic>Psp<\/jats:italic>\n                          <jats:styled-content style=\"fixed-case\">NPS<\/jats:styled-content>\n                          3121\u2010 and\/or flg22\u2010induced defense responses including defense gene expression, callose deposition, and reactive oxygen species (\n                          <jats:styled-content style=\"fixed-case\">ROS<\/jats:styled-content>\n                          ) and salicylic acid (\n                          <jats:styled-content style=\"fixed-case\">SA<\/jats:styled-content>\n                          ) accumulation. Mutations in Elongator also attenuate the\n                          <jats:styled-content style=\"fixed-case\">ROS<\/jats:styled-content>\n                          \u2013\n                          <jats:styled-content style=\"fixed-case\">SA<\/jats:styled-content>\n                          amplification loop. We show that suppressed\n                          <jats:styled-content style=\"fixed-case\">ROS<\/jats:styled-content>\n                          and\n                          <jats:styled-content style=\"fixed-case\">SA<\/jats:styled-content>\n                          accumulation in Elongator mutants is correlated with reduced expression of the\n                          <jats:italic>Arabidopsis respiratory burst oxidase homologue AtrbohD<\/jats:italic>\n                          and the\n                          <jats:styled-content style=\"fixed-case\">SA<\/jats:styled-content>\n                          biosynthesis gene\n                          <jats:italic>ISOCHORISMATE SYNTHASE1<\/jats:italic>\n                          (\n                          <jats:italic>ICS1<\/jats:italic>\n                          ). Furthermore, we found that the Elongator subunit\n                          <jats:styled-content style=\"fixed-case\">ELP<\/jats:styled-content>\n                          2 is associated with the chromatin of\n                          <jats:italic>AtrbohD<\/jats:italic>\n                          and\n                          <jats:italic>\n                            <jats:styled-content style=\"fixed-case\">ICS<\/jats:styled-content>\n                            1\n                          <\/jats:italic>\n                          and is required for maintaining basal histone H3 acetylation levels in these key defense genes.\n                        <\/jats:p>\n                      <\/jats:list-item>\n                      <jats:list-item>\n                        <jats:p>\n                          As both\n                          <jats:italic>AtrbohD<\/jats:italic>\n                          and\n                          <jats:italic>\n                            <jats:styled-content style=\"fixed-case\">ICS<\/jats:styled-content>\n                            1\n                          <\/jats:italic>\n                          contribute to nonhost resistance against\n                          <jats:italic>Xcc<\/jats:italic>\n                          , our results reveal an epigenetic mechanism by which Elongator regulates nonhost resistance in Arabidopsis.\n                        <\/jats:p>\n                      <\/jats:list-item>\n                    <\/jats:list>\n                  <\/jats:p>","DOI":"10.1111\/nph.14442","type":"journal-article","created":{"date-parts":[[2017,1,30]],"date-time":"2017-01-30T06:50:12Z","timestamp":1485759012000},"page":"1245-1259","update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":28,"title":["The Arabidopsis Elongator complex is required for nonhost resistance against the bacterial pathogens\n                    <i>Xanthomonas citri<\/i>\n          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