{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,8,23]],"date-time":"2026-08-23T16:26:05Z","timestamp":1787502365363,"version":"build-2736575974"},"reference-count":23,"publisher":"Wiley","issue":"3","license":[{"start":{"date-parts":[[2013,5,9]],"date-time":"2013-05-09T00:00:00Z","timestamp":1368057600000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Oral Diseases"],"published-print":{"date-parts":[[2014,4]]},"abstract":"<jats:sec>\n                    <jats:title>Objective<\/jats:title>\n                    <jats:p>\n                      To determine the frequency of\n                      <jats:italic>\n                        <jats:styled-content style=\"fixed-case\">C<\/jats:styled-content>\n                        andida\n                      <\/jats:italic>\n                      spp., xerostomia, and salivary flow rate (\n                      <jats:styled-content style=\"fixed-case\">SFR<\/jats:styled-content>\n                      ) in three different groups: patients with\n                      <jats:styled-content style=\"fixed-case\">OLP<\/jats:styled-content>\n                      (\n                      <jats:styled-content style=\"fixed-case\">OLP<\/jats:styled-content>\n                      group), patients with oral mucosal lesions other than\n                      <jats:styled-content style=\"fixed-case\">OLP<\/jats:styled-content>\n                      (non\u2010\n                      <jats:styled-content style=\"fixed-case\">OLP<\/jats:styled-content>\n                      group), and subjects without oral mucosal lesions (control group).\n                    <\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Material and methods<\/jats:title>\n                    <jats:p>\n                      Xerostomia as well as\n                      <jats:styled-content style=\"fixed-case\">SFR<\/jats:styled-content>\n                      was investigated in the three groups. Samples for isolation of\n                      <jats:italic>\n                        <jats:styled-content style=\"fixed-case\">C<\/jats:styled-content>\n                        andida\n                      <\/jats:italic>\n                      spp. were collected from\n                      <jats:styled-content style=\"fixed-case\">OLP<\/jats:styled-content>\n                      lesions (38 patients), non\u2010\n                      <jats:styled-content style=\"fixed-case\">OLP<\/jats:styled-content>\n                      lesions (28 patients), and healthy subjects (32 subjects).\n                    <\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Results<\/jats:title>\n                    <jats:p>\n                      There was no statistically significant difference regarding the frequency of xerostomia and hyposalivation among the three groups (\n                      <jats:italic>P<\/jats:italic>\n                      \u00a0&gt;\u00a00.05). A higher prevalence for colonization by\n                      <jats:italic>\n                        <jats:styled-content style=\"fixed-case\">C<\/jats:styled-content>\n                        andida\n                      <\/jats:italic>\n                      spp. was found in the healthy subject as compared to that of patients with\n                      <jats:styled-content style=\"fixed-case\">OLP<\/jats:styled-content>\n                      (\n                      <jats:italic>P\u00a0<\/jats:italic>\n                      =\u00a00.03) and non\u2010\n                      <jats:styled-content style=\"fixed-case\">OLP<\/jats:styled-content>\n                      (\n                      <jats:italic>P\u00a0<\/jats:italic>\n                      =\u00a00.02) groups. Low\n                      <jats:styled-content style=\"fixed-case\">SFR<\/jats:styled-content>\n                      was not a factor for colonization by\n                      <jats:italic>\n                        <jats:styled-content style=\"fixed-case\">C<\/jats:styled-content>\n                        andida\n                      <\/jats:italic>\n                      spp.\n                    <\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Conclusions<\/jats:title>\n                    <jats:p>\n                      Xerostomia and hyposalivation occur with similar frequency in subjects with and without oral lesions; also, the presence of oral lesions does not increase the susceptibility to colonization by\n                      <jats:italic>\n                        <jats:styled-content style=\"fixed-case\">C<\/jats:styled-content>\n                        andida\n                      <\/jats:italic>\n                      spp. It seems that any study implicating\n                      <jats:italic>\n                        <jats:styled-content style=\"fixed-case\">C<\/jats:styled-content>\n                        andida\n                      <\/jats:italic>\n                      spp. in the malignant transformation of oral lesions should be carried out mostly on a biochemical basis, that is, by testing the capability of\n                      <jats:italic>\n                        <jats:styled-content style=\"fixed-case\">C<\/jats:styled-content>\n                        andida\n                      <\/jats:italic>\n                      spp. to produce carcinogenic enzyme.\n                    <\/jats:p>\n                  <\/jats:sec>","DOI":"10.1111\/odi.12120","type":"journal-article","created":{"date-parts":[[2013,4,19]],"date-time":"2013-04-19T10:29:49Z","timestamp":1366367389000},"source":"Crossref","is-referenced-by-count":37,"title":["Prevalence of\n                    <i>\n                      <scp>C<\/scp>\n                      andida\n                    <\/i>\n                    spp., xerostomia, and hyposalivation in oral lichen planus \u2013 A controlled study"],"prefix":"10.1111","volume":"20","author":[{"given":"G","family":"Artico","sequence":"first","affiliation":[{"name":"Department of Stomatology School of Dentistry University of S\u00e3o Paulo S\u00e3o Paulo Brazil"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"RS","family":"Freitas","sequence":"additional","affiliation":[{"name":"Division of Dermatology Laboratory of Medical Investigation (LIM\/53) School of Medicine University of S\u00e3o Paulo S\u00e3o Paulo Brazil"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"AM","family":"Santos Filho","sequence":"additional","affiliation":[{"name":"Division of Dermatology Laboratory of Medical Investigation (LIM\/53) School of Medicine University of S\u00e3o Paulo S\u00e3o Paulo Brazil"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"G","family":"Benard","sequence":"additional","affiliation":[{"name":"Division of Dermatology Laboratory of Medical Investigation (LIM\/53) School of Medicine University of S\u00e3o Paulo S\u00e3o Paulo Brazil"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"R","family":"Romiti","sequence":"additional","affiliation":[{"name":"Division of Dermatology School of Medicine University of S\u00e3o Paulo S\u00e3o Paulo Brazil"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"DA","family":"Migliari","sequence":"additional","affiliation":[{"name":"Department of Stomatology School of Dentistry University of S\u00e3o Paulo S\u00e3o Paulo 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