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We used ICR mice as non\u2010permissive hosts, divided them into groups and experimentally infected them with infective larvae of the\n                    <jats:styled-content style=\"fixed-case\">\n                      <jats:italic>Angiostrongylus cantonensis<\/jats:italic>\n                    <\/jats:styled-content>\n                    . Two groups of infected mice were injected intraperitoneally with either mouse IL\u201025 or an anti\u2010IL\u201025 monoclonal antibody (mAb) at 3\u2009days post\u2010infection (dpi), followed by booster injections at the same dose every 5\u2009days. Serum samples and brain tissues were collected weekly from each group for immunological and pathological examinations. The IL\u201025\u2010treated group exhibited significant increases in eosinophil percentages and levels of immunoglobulin E (IgE), as well as in the levels of IL\u20105 and IL\u201013. The severity of eosinophilic meningitis was exacerbated in the IL\u201025\u2010treated group 21\u2009dpi. These results suggest that IL\u201025 may enhance eosinophil\u2010associated inflammation in mice infected with\n                    <jats:styled-content style=\"fixed-case\">\n                      <jats:italic>A. cantonensis<\/jats:italic>\n                    <\/jats:styled-content>\n                    .\n                  <\/jats:p>","DOI":"10.1111\/pim.70088","type":"journal-article","created":{"date-parts":[[2026,6,8]],"date-time":"2026-06-08T00:35:01Z","timestamp":1780878901000},"update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":0,"title":["<scp>IL<\/scp>\n                    \u201025 Enhances Eosinophil\u2010Associated Inflammation in\n                    <scp>\n                      <i>Angiostrongylus cantonensis<\/i>\n                    <\/scp>\n                    \u2010Infected 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