{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,10,27]],"date-time":"2026-10-27T05:16:56Z","timestamp":1793078216111,"version":"4.5.0"},"reference-count":34,"publisher":"Wiley","issue":"2","license":[{"start":{"date-parts":[[2013,2,4]],"date-time":"2013-02-04T00:00:00Z","timestamp":1359936000000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Paediatric Perinatal Epid"],"published-print":{"date-parts":[[2013,3]]},"abstract":"<jats:title>Abstract<\/jats:title><jats:sec><jats:title>Background<\/jats:title><jats:p>Infants born preterm account for a substantial part of neonatal morbidity, with acute respiratory disorders being a dominating clinical problem. Whereas focus in recent studies has been on extremely and very preterm infants, less is known about contemporary rates and risk factors for acute respiratory morbidity in moderately and late preterm infants. The objective of this population\u2010based <jats:styled-content style=\"fixed-case\">S<\/jats:styled-content>wedish study was to establish rates for different acute respiratory diseases in moderately preterm infants, and to identify maternal, obstetric and neonatal risk factors for the two most common diagnoses, transient tachypnoea of the newborn (<jats:styled-content style=\"fixed-case\">TTN<\/jats:styled-content>) and respiratory distress syndrome (<jats:styled-content style=\"fixed-case\">RDS<\/jats:styled-content>).<\/jats:p><\/jats:sec><jats:sec><jats:title>Methods<\/jats:title><jats:p>The study included 4679 moderately preterm [gestational age (<jats:styled-content style=\"fixed-case\">GA<\/jats:styled-content>): 30 to 34 weeks], 15\u2009036 late preterm infants (<jats:styled-content style=\"fixed-case\">GA<\/jats:styled-content> 35 to 36 weeks) and 451\u2009479 term infants (<jats:styled-content style=\"fixed-case\">GA<\/jats:styled-content>: 37 to 41 weeks). All infants were born in 2004\u20132008.<\/jats:p><\/jats:sec><jats:sec><jats:title>Results<\/jats:title><jats:p>In moderately preterm infants, risk factors for <jats:styled-content style=\"fixed-case\">TTN<\/jats:styled-content> in multivariable analyses were multiparity, caesarean section before and after onset of labour, male sex, <jats:styled-content style=\"fixed-case\">A<\/jats:styled-content>pgar score 4\u20136 at 5\u2009min and lower <jats:styled-content style=\"fixed-case\">GA<\/jats:styled-content>. Risk factors for <jats:styled-content style=\"fixed-case\">RDS<\/jats:styled-content> were multiparity, caesarean section before and after onset of labour, male sex, Apgar score &lt;7 at 5\u2009min and lower <jats:styled-content style=\"fixed-case\">GA<\/jats:styled-content>. Preterm rupture of membranes, antenatal corticosteroid treatment and being small for gestational age reduced the risk of <jats:styled-content style=\"fixed-case\">RDS<\/jats:styled-content>.<\/jats:p><\/jats:sec><jats:sec><jats:title>Conclusion<\/jats:title><jats:p>We conclude that acute respiratory morbidity in moderately preterm infants is common and predicted by multiparity, caesarean section, low <jats:styled-content style=\"fixed-case\">A<\/jats:styled-content>pgar score and male sex.<\/jats:p><\/jats:sec>","DOI":"10.1111\/ppe.12035","type":"journal-article","created":{"date-parts":[[2013,10,15]],"date-time":"2013-10-15T13:22:21Z","timestamp":1381843341000},"page":"172-181","source":"Crossref","is-referenced-by-count":62,"title":["Risk Factors for Acute Respiratory Morbidity in Moderately Preterm Infants"],"prefix":"10.1111","volume":"27","author":[{"given":"Maria","family":"Altman","sequence":"first","affiliation":[{"name":"Department of Clinical Science, Intervention and Technology Karolinska Institutet  Stockholm Sweden"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Mireille","family":"Vanp\u00e9e","sequence":"additional","affiliation":[{"name":"Department of Women's and Children's Health Karolinska Institutet  Stockholm Sweden"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Sven","family":"Cnattingius","sequence":"additional","affiliation":[{"name":"Clinical Epidemiology Unit Department of Medicine Karolinska Institutet  Stockholm Sweden"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Mikael","family":"Norman","sequence":"additional","affiliation":[{"name":"Department of Clinical Science, Intervention and Technology Karolinska Institutet  Stockholm Sweden"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"311","published-online":{"date-parts":[[2013,2,4]]},"reference":[{"key":"e_1_2_5_2_1","doi-asserted-by":"publisher","DOI":"10.1016\/S0140-6736(08)60074-4"},{"key":"e_1_2_5_3_1","doi-asserted-by":"publisher","DOI":"10.1016\/j.jpeds.2010.07.047"},{"key":"e_1_2_5_4_1","doi-asserted-by":"publisher","DOI":"10.1097\/AOG.0b013e3181af6931"},{"key":"e_1_2_5_5_1","doi-asserted-by":"publisher","DOI":"10.1542\/peds.2006-3629"},{"key":"e_1_2_5_6_1","doi-asserted-by":"publisher","DOI":"10.1542\/peds.2006-3260"},{"key":"e_1_2_5_7_1","doi-asserted-by":"publisher","DOI":"10.1097\/01.ANC.0000267913.58726.f3"},{"key":"e_1_2_5_8_1","doi-asserted-by":"publisher","DOI":"10.1542\/peds.2008-1654"},{"key":"e_1_2_5_9_1","doi-asserted-by":"publisher","DOI":"10.1111\/j.1651-2227.1981.tb06228.x"},{"key":"e_1_2_5_10_1","doi-asserted-by":"publisher","DOI":"10.1111\/j.1651-2227.2010.01846.x"},{"key":"e_1_2_5_11_1","doi-asserted-by":"publisher","DOI":"10.1016\/j.ajog.2010.08.021"},{"key":"e_1_2_5_12_1","doi-asserted-by":"publisher","DOI":"10.1001\/jama.2009.771"},{"key":"e_1_2_5_13_1","doi-asserted-by":"publisher","DOI":"10.1136\/adc.2005.087031"},{"key":"e_1_2_5_14_1","doi-asserted-by":"publisher","DOI":"10.1038\/jp.2011.155"},{"key":"e_1_2_5_15_1","doi-asserted-by":"publisher","DOI":"10.1111\/j.1651-2227.1996.tb14164.x"},{"key":"e_1_2_5_16_1","unstructured":"Socialstyrelsen. 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