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This study aims to assess racial\/ethnic differences in relative contribution of risk factors of gestational diabetes mellitus (<jats:styled-content style=\"fixed-case\">GDM<\/jats:styled-content>) among <jats:styled-content style=\"fixed-case\">A<\/jats:styled-content>sian subgroups (<jats:styled-content style=\"fixed-case\">A<\/jats:styled-content>sian <jats:styled-content style=\"fixed-case\">I<\/jats:styled-content>ndian, <jats:styled-content style=\"fixed-case\">C<\/jats:styled-content>hinese, <jats:styled-content style=\"fixed-case\">F<\/jats:styled-content>ilipino, <jats:styled-content style=\"fixed-case\">J<\/jats:styled-content>apanese, <jats:styled-content style=\"fixed-case\">K<\/jats:styled-content>orean, and <jats:styled-content style=\"fixed-case\">V<\/jats:styled-content>ietnamese), Hispanics, non\u2010Hispanic blacks, and non\u2010Hispanic whites.<\/jats:p><\/jats:sec><jats:sec><jats:title>Methods<\/jats:title><jats:p>Pregnant women in 2007\u20132012 were identified through <jats:styled-content style=\"fixed-case\">C<\/jats:styled-content>alifornia state birth certificate records and linked to the electronic health records in a large mixed\u2010payer ambulatory care organisation in <jats:styled-content style=\"fixed-case\">N<\/jats:styled-content>orthern <jats:styled-content style=\"fixed-case\">C<\/jats:styled-content>alifornia (<jats:italic>n<\/jats:italic>\u2009=\u200924\u2009195). Relative risk and population attributable fraction (<jats:styled-content style=\"fixed-case\">PAF<\/jats:styled-content>) for specific racial\/ethnic groups were calculated to assess the contributions of advanced maternal age, overweight\/obesity (<jats:styled-content style=\"fixed-case\">C<\/jats:styled-content>enters for <jats:styled-content style=\"fixed-case\">D<\/jats:styled-content>isease <jats:styled-content style=\"fixed-case\">C<\/jats:styled-content>ontrol and Prevention (CDC) standards and <jats:styled-content style=\"fixed-case\">W<\/jats:styled-content>orld <jats:styled-content style=\"fixed-case\">H<\/jats:styled-content>ealth <jats:styled-content style=\"fixed-case\">O<\/jats:styled-content>rganization (<jats:styled-content style=\"fixed-case\">WHO<\/jats:styled-content>)\/<jats:styled-content style=\"fixed-case\">A<\/jats:styled-content>merican <jats:styled-content style=\"fixed-case\">D<\/jats:styled-content>iabetes <jats:styled-content style=\"fixed-case\">A<\/jats:styled-content>ssociation (<jats:styled-content style=\"fixed-case\">ADA<\/jats:styled-content>) body mass index cut\u2010offs for <jats:styled-content style=\"fixed-case\">A<\/jats:styled-content>sians), family history of type 2 diabetes, and foreign\u2010born status.<\/jats:p><\/jats:sec><jats:sec><jats:title>Results<\/jats:title><jats:p><jats:styled-content style=\"fixed-case\">GDM<\/jats:styled-content> was most prevalent among <jats:styled-content style=\"fixed-case\">A<\/jats:styled-content>sian <jats:styled-content style=\"fixed-case\">I<\/jats:styled-content>ndians (19.3%). Relative risks were similar across all race\/ethnic groups. Advanced maternal age had higher <jats:styled-content style=\"fixed-case\">PAFs<\/jats:styled-content> in non\u2010<jats:styled-content style=\"fixed-case\">H<\/jats:styled-content>ispanic whites (22.5%) and Hispanics (22.7%). Meanwhile family history (<jats:styled-content style=\"fixed-case\">A<\/jats:styled-content>sian <jats:styled-content style=\"fixed-case\">I<\/jats:styled-content>ndians 22.6%, <jats:styled-content style=\"fixed-case\">C<\/jats:styled-content>hinese 22.9%) and foreign\u2010borne status (<jats:styled-content style=\"fixed-case\">C<\/jats:styled-content>hinese 40.2%, <jats:styled-content style=\"fixed-case\">F<\/jats:styled-content>ilipinos 30.2%) had higher <jats:styled-content style=\"fixed-case\">PAFs<\/jats:styled-content> in <jats:styled-content style=\"fixed-case\">A<\/jats:styled-content>sian subgroups. Overweight\/obesity was the most important <jats:styled-content style=\"fixed-case\">GDM<\/jats:styled-content> risk factor for non\u2010<jats:styled-content style=\"fixed-case\">H<\/jats:styled-content>ispanic whites, <jats:styled-content style=\"fixed-case\">H<\/jats:styled-content>ispanics, <jats:styled-content style=\"fixed-case\">A<\/jats:styled-content>sian <jats:styled-content style=\"fixed-case\">I<\/jats:styled-content>ndians, and <jats:styled-content style=\"fixed-case\">F<\/jats:styled-content>ilipinos when the <jats:styled-content style=\"fixed-case\">WHO<\/jats:styled-content>\/<jats:styled-content style=\"fixed-case\">ADA<\/jats:styled-content> cut\u2010off points were applied. Advanced maternal age was the only risk factor studied that was modified by race\/ethnicity, with non\u2010<jats:styled-content style=\"fixed-case\">H<\/jats:styled-content>ispanic white and <jats:styled-content style=\"fixed-case\">H<\/jats:styled-content>ispanic women being more adversely affected than other racial\/ethnic groups.<\/jats:p><\/jats:sec><jats:sec><jats:title>Conclusions<\/jats:title><jats:p>Overweight\/obesity, advanced maternal age, family history of type 2 diabetes, and foreign\u2010borne status are important risk factors for <jats:styled-content style=\"fixed-case\">GDM<\/jats:styled-content>. The relative contributions of these risk factors differ by race\/ethnicity, mainly due to differences in population prevalence of these risk factors.<\/jats:p><\/jats:sec>","DOI":"10.1111\/ppe.12209","type":"journal-article","created":{"date-parts":[[2015,7,22]],"date-time":"2015-07-22T16:54:49Z","timestamp":1437584089000},"page":"436-443","source":"Crossref","is-referenced-by-count":158,"title":["Racial\/Ethnic Differences in Gestational Diabetes Prevalence and Contribution of Common Risk Factors"],"prefix":"10.1111","volume":"29","author":[{"given":"Jia","family":"Pu","sequence":"first","affiliation":[{"name":"Research Institute Palo Alto Medical Foundation  Palo Alto CA"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Beinan","family":"Zhao","sequence":"additional","affiliation":[{"name":"Research Institute Palo Alto Medical Foundation  Palo Alto CA"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Elsie J.","family":"Wang","sequence":"additional","affiliation":[{"name":"School of Medicine Stanford University School of Medicine  Stanford CA"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Vani","family":"Nimbal","sequence":"additional","affiliation":[{"name":"Research Institute Palo Alto Medical Foundation  Palo Alto CA"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Sarah","family":"Osmundson","sequence":"additional","affiliation":[{"name":"Lucile Packard Children's Hospital Stanford University School of Medicine  Stanford CA"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Liza","family":"Kunz","sequence":"additional","affiliation":[{"name":"Research Institute Palo Alto Medical Foundation  Palo Alto CA"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Rita A.","family":"Popat","sequence":"additional","affiliation":[{"name":"Division of Epidemiology Department of Health Research and Policy School of Medicine Stanford University  Stanford CA"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Sukyung","family":"Chung","sequence":"additional","affiliation":[{"name":"Research Institute Palo Alto Medical Foundation  Palo Alto CA"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Latha P.","family":"Palaniappan","sequence":"additional","affiliation":[{"name":"School of Medicine Stanford University School of Medicine  Stanford CA"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"311","published-online":{"date-parts":[[2015,7,22]]},"reference":[{"key":"e_1_2_7_2_1","doi-asserted-by":"publisher","DOI":"10.5888\/pcd11.130415"},{"key":"e_1_2_7_3_1","doi-asserted-by":"publisher","DOI":"10.2337\/dc07-s206"},{"key":"e_1_2_7_4_1","doi-asserted-by":"publisher","DOI":"10.1016\/S0140-6736(09)60731-5"},{"key":"e_1_2_7_5_1","doi-asserted-by":"publisher","DOI":"10.2337\/dc06-1816"},{"key":"e_1_2_7_6_1","doi-asserted-by":"publisher","DOI":"10.2337\/diacare.25.10.1862"},{"key":"e_1_2_7_7_1","doi-asserted-by":"publisher","DOI":"10.2337\/dc11-S062"},{"issue":"2","key":"e_1_2_7_8_1","first-page":"B9","article-title":"Epidemiology of glucose intolerance and gestational diabetes in women of childbearing age","volume":"21","author":"King H","year":"1998","journal-title":"Diabetes Care"},{"key":"e_1_2_7_9_1","doi-asserted-by":"publisher","DOI":"10.1001\/jama.1997.03550130052036"},{"key":"e_1_2_7_10_1","doi-asserted-by":"publisher","DOI":"10.1016\/j.whi.2010.06.003"},{"key":"e_1_2_7_11_1","doi-asserted-by":"publisher","DOI":"10.2337\/diacare.28.3.579"},{"key":"e_1_2_7_12_1","doi-asserted-by":"publisher","DOI":"10.2105\/AJPH.2005.066100"},{"key":"e_1_2_7_13_1","doi-asserted-by":"publisher","DOI":"10.2105\/AJPH.2013.301469"},{"key":"e_1_2_7_14_1","doi-asserted-by":"publisher","DOI":"10.2337\/dc11-2267"},{"key":"e_1_2_7_15_1","doi-asserted-by":"publisher","DOI":"10.1111\/j.1365-3016.2010.01140.x"},{"key":"e_1_2_7_16_1","doi-asserted-by":"publisher","DOI":"10.1016\/S0140-6736(03)15268-3"},{"key":"e_1_2_7_17_1","doi-asserted-by":"publisher","DOI":"10.2337\/dc14-2391"},{"key":"e_1_2_7_18_1","unstructured":"California Diabetes and Pregnancy Program.Guidelines for diagnosis of hyperglycemia in pregnancy2012; 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