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As such, interleukin\u201017 can mediate protective innate immunity to pathogens or contribute to the pathogenesis of inflammatory diseases, such as psoriasis and rheumatoid arthritis. This review summarizes the basic biology of interleukin\u201017 and discusses its emerging role in periodontal disease. The current burden of evidence from human and animal model studies suggests that the net effect of interleukin\u201017 signaling promotes disease development. In addition to promoting neutrophilic inflammation, interleukin\u201017 has potent pro\u2010osteoclastogenic effects that are likely to contribute to the pathogenesis of periodontitis, rheumatoid arthritis and other diseases involving bone immunopathology. Systemic treatments with anti\u2010interleukin\u201017 biologics have shown promising results in clinical trials for psoriasis and rheumatoid arthritis; however, their impact on the highly prevalent periodontal disease has not been investigated or reported. Future clinical trials, preferably using locally administered interleukin\u201017 blockers, are required to implicate conclusivelyinterleukin\u201017 in periodontitis and, more importantly, to establish an effective adjunctive treatment for this oral inflammatory disease.<\/jats:p>","DOI":"10.1111\/prd.12083","type":"journal-article","created":{"date-parts":[[2015,8,7]],"date-time":"2015-08-07T21:04:16Z","timestamp":1438981456000},"page":"142-159","source":"Crossref","is-referenced-by-count":436,"title":["Basic biology and role of interleukin\u201017 in immunity and 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