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In this study, we show that, in addition to previously described longevity phenotypes, constitutive overexpression of\n                    <jats:italic>AHL15<\/jats:italic>\n                    in Arabidopsis delays leaf senescence, whereas\n                    <jats:italic>ahl15<\/jats:italic>\n                    loss of function accelerates this process. Dexamethasone\u2010induced nuclear localization of AHL15\u2010GR during dark\u2010triggered senescence results in a stay\u2010green phenotype and represses the expression of several early senescence\u2010associated genes. Among these, the\n                    <jats:italic>ORESARA1 (ORE1)<\/jats:italic>\n                    locus is directly bound by AHL15, suggesting a direct delaying effect of AHL15 on senescence. Furthermore, we demonstrate that AHL15 acts by directly repressing the expression of several\n                    <jats:italic>CYTOKININ OXIDASE (CKX)<\/jats:italic>\n                    genes involved in cytokinin inactivation, resulting in delayed degradation of cytokinins during dark\u2010induced senescence. Cytokinins are known to inhibit senescence, and this, together with the downregulation of\n                    <jats:italic>ORE1<\/jats:italic>\n                    expression, explains the delaying effect of AHL15 on senescence.\n                  <\/jats:p>","DOI":"10.1111\/tpj.71177","type":"journal-article","created":{"date-parts":[[2026,10,15]],"date-time":"2026-10-15T05:45:07Z","timestamp":1792043107000},"update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":0,"title":["The plant longevity gene\n                    <i>AHL15<\/i>\n                    delays leaf senescence by repressing\n                    <i>ORESARA1<\/i>\n                    and cytokinin degradation"],"prefix":"10.1111","volume":"128","author":[{"ORCID":"https:\/\/orcid.org\/0009-0007-3029-2058","authenticated-orcid":false,"given":"Thalia","family":"Luden","sequence":"first","affiliation":[{"name":"Plant Developmental Genetics Institute of Biology Leiden, 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