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We show that the influence of model parameters on the total concentrations of amyloid precursor protein (APP) and tubulin-associated unit (tau) protein in the axon is reciprocal to the influence of the same parameters on the average velocities of the same proteins during their transport in the axon. The results of our analysis also suggest that in the beginning of AD the aggregation of amyloid-\u03b2 and misfolded tau protein have little effect on transport of APP and tau in the axon, which suggests that early damage in AD may be reversible.<\/jats:p>","DOI":"10.1115\/1.4041905","type":"journal-article","created":{"date-parts":[[2018,11,1]],"date-time":"2018-11-01T15:32:57Z","timestamp":1541086377000},"update-policy":"https:\/\/doi.org\/10.1115\/crossmarkpolicy-asme","source":"Crossref","is-referenced-by-count":5,"title":["A Numerical Study of Sensitivity Coefficients for a Model of Amyloid Precursor Protein and Tubulin-Associated Unit Protein Transport and Agglomeration in Neurons at the Onset of Alzheimer's Disease"],"prefix":"10.1115","volume":"141","author":[{"given":"I. 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