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Thus, TCR of oxidative damage requires an XPG function distinct from its NER endonuclease activity. These results raise the possibility that defective TCR of oxidative damage contributes to the developmental defects associated with CS.<\/jats:p>","DOI":"10.1126\/science.275.5302.990","type":"journal-article","created":{"date-parts":[[2002,7,27]],"date-time":"2002-07-27T09:45:15Z","timestamp":1027763115000},"page":"990-993","source":"Crossref","is-referenced-by-count":243,"title":["Defective Transcription-Coupled Repair of Oxidative Base Damage in Cockayne Syndrome Patients from XP Group G"],"prefix":"10.1126","volume":"275","author":[{"given":"Priscilla K.","family":"Cooper","sequence":"first","affiliation":[{"name":"P. K. 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