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Traunm\u00fcller\n                    <jats:italic>et al.<\/jats:italic>\n                    found that the RNA-binding protein SLM2 regulated a very small number of alternative splicing decisions in mouse hippocampal neurons. Disrupting this splicing program resulted in specific defects in transsynaptic protein complexes. Glutamatergic transmission and synaptic plasticity were impaired. Genetically restoring one single alternative splicing event was enough to recover synaptic plasticity. SLM2 thus activates a highly specific alternative splicing program that controls the properties of glutamatergic synapses.\n                  <\/jats:p>\n                  <jats:p>\n                    <jats:italic>Science<\/jats:italic>\n                    , this issue p.\n                    <jats:related-article xmlns:xlink=\"http:\/\/www.w3.org\/1999\/xlink\" ext-link-type=\"doi\" issue=\"6288\" page=\"982\" related-article-type=\"in-this-issue\" vol=\"352\" xlink:href=\"10.1126\/science.aaf2397\">982<\/jats:related-article>\n                  <\/jats:p>","DOI":"10.1126\/science.aaf2397","type":"journal-article","created":{"date-parts":[[2016,5,12]],"date-time":"2016-05-12T23:09:07Z","timestamp":1463094547000},"page":"982-986","source":"Crossref","is-referenced-by-count":148,"title":["Control of neuronal synapse specification by a highly dedicated alternative splicing 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