{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,11,7]],"date-time":"2026-11-07T19:55:54Z","timestamp":1794081354951,"version":"4.8.0"},"reference-count":40,"publisher":"American Society for Microbiology","issue":"7","license":[{"start":{"date-parts":[[2020,6,23]],"date-time":"2020-06-23T00:00:00Z","timestamp":1592870400000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/journals.asm.org\/non-commercial-tdm-license"}],"funder":[{"name":"Otsuka Pharmaceutical Co. 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Outbreaks of\n            <jats:named-content content-type=\"genus-species\">C. difficile<\/jats:named-content>\n            infection (CDI) have been caused by strains with acquired antimicrobial resistance, particularly fluoroquinolone resistance, including\n            <jats:named-content content-type=\"genus-species\">C. difficile<\/jats:named-content>\n            ribotype (RT) 027 in North America and Europe and RT 017, the most common strain in Asia. 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