{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2023,9,13]],"date-time":"2023-09-13T16:39:56Z","timestamp":1694623196336},"reference-count":13,"publisher":"American Society for Microbiology","issue":"8","license":[{"start":{"date-parts":[[2004,8,1]],"date-time":"2004-08-01T00:00:00Z","timestamp":1091318400000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/journals.asm.org\/non-commercial-tdm-license"}],"content-domain":{"domain":["journals.asm.org"],"crossmark-restriction":true},"short-container-title":["Antimicrob Agents Chemother"],"published-print":{"date-parts":[[2004,8]]},"abstract":"<jats:title>ABSTRACT<\/jats:title>\n          <jats:p>\n            Commonly used \u201cinactive\u201d pharmaceutical excipients were tested in a previously developed minimum cidal concentration assay to assess their ability to kill\n            <jats:italic>Chlamydia trachomatis<\/jats:italic>\n            topically. Sixteen excipients were evaluated in these studies under various conditions. A range of activities was found among the excipients that could be tested in our assay system.\n          <\/jats:p>","DOI":"10.1128\/aac.48.8.3200-3202.2004","type":"journal-article","created":{"date-parts":[[2004,7,23]],"date-time":"2004-07-23T18:45:20Z","timestamp":1090608320000},"page":"3200-3202","update-policy":"http:\/\/dx.doi.org\/10.1128\/asmj-crossmark-policy-page","source":"Crossref","is-referenced-by-count":3,"title":["Susceptibility of\n            <i>Chlamydia trachomatis<\/i>\n            to Excipients Commonly Used in Topical Microbicide Formulations"],"prefix":"10.1128","volume":"48","author":[{"given":"M. F.","family":"Lampe","sequence":"first","affiliation":[{"name":"Department of Laboratory Medicine"},{"name":"Division of Infectious Diseases, University of Washington, Seattle, Washington"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"L. C.","family":"Rohan","sequence":"additional","affiliation":[{"name":"Magee-Womens Research Institute, Pittsburgh, Pennsylvania"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"M. C.","family":"Skinner","sequence":"additional","affiliation":[{"name":"Department of Laboratory Medicine"},{"name":"Division of Infectious Diseases, University of Washington, Seattle, Washington"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"W. E.","family":"Stamm","sequence":"additional","affiliation":[{"name":"Division of Infectious Diseases, University of Washington, Seattle, Washington"}],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"235","reference":[{"key":"e_1_3_2_2_2","doi-asserted-by":"publisher","DOI":"10.1128\/AAC.46.1.34-41.2002"},{"key":"e_1_3_2_3_2","doi-asserted-by":"crossref","first-page":"685","DOI":"10.1093\/jac\/46.5.685","volume":"46","year":"2000","unstructured":"Belec, L., C. Tevi-Benissan, A. Bianchi, S. Citigny, M. Beumont-Mauviel, A. Si-Mohamed, and J. E. Malkin. 2000. In vitro inactivation of Chlamydia trachomatis and a panel of DNA (HSV-2, CMV, adenovirus, BK virus) and RNA (RSV, enterovirus) viruses by the spermicide benzalkonium chloride. J. Antimicrob. Chemother.46:685-693.","journal-title":"J. Antimicrob. Chemother."},{"key":"e_1_3_2_4_2","doi-asserted-by":"crossref","first-page":"237","DOI":"10.1034\/j.1399-3011.2003.00053.x","volume":"61","year":"2003","unstructured":"Chong-Cerrillo, C., M. E. Selsted, E. M. Peterson, and L. M. de la Maza. 2003. Susceptibility of human and murine Chlamydia trachomatis serovars to granulocyte- and epithelium-derived antimicrobial peptides. J. Peptide Res.61:237-242.","journal-title":"J. Peptide Res."},{"key":"e_1_3_2_5_2","first-page":"14","volume":"25","year":"2001","unstructured":"Garg, S., K. R. Tambwekar, K. Vermani, A. Garg, C. L. Kaul, and L. J. D. Zaneveld. 2001. Compendium of pharmaceutical excipients for vaginal formulations. Pharm. Technol.25:14-24. 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Handbook of pharmaceutical excipients 3rd ed. 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