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Using\n            <jats:named-content content-type=\"genus-species\">Helicobacter pylori<\/jats:named-content>\n            infection as a model for chronic mucosal infection, we previously published that IL-21 is required for the development of gastritis in response to infection. Concomitant with protection from chronic inflammation,\n            <jats:named-content content-type=\"genus-species\">H. pylori<\/jats:named-content>\n            -infected IL-21\n            <jats:sup>\u2212\/\u2212<\/jats:sup>\n            mice exhibited limited Th1 and Th17 responses in their gastric mucosa.\n          <\/jats:p>","DOI":"10.1128\/iai.00237-19","type":"journal-article","created":{"date-parts":[[2019,8,1]],"date-time":"2019-08-01T13:25:00Z","timestamp":1564665900000},"update-policy":"https:\/\/doi.org\/10.1128\/asmj-crossmark-policy-page","source":"Crossref","is-referenced-by-count":12,"title":["Interleukin-21 (IL-21) Downregulates Dendritic Cell Cytokine Responses to Helicobacter pylori and Modulates T Lymphocyte IL-17A Expression in Peyer\u2019s Patches during Infection"],"prefix":"10.1128","volume":"87","author":[{"given":"Sharia","family":"Yasmin","sequence":"first","affiliation":[{"name":"Department of Medicine, Division of Infectious Disease, Vanderbilt University School of Medicine, Vanderbilt University Medical Center, Nashville, Tennessee, USA"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Beverly R. 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