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It is currently unclear how latency is established in these cells\n            <jats:italic>in vivo<\/jats:italic>\n            . We show that in CD4\n            <jats:sup>+<\/jats:sup>\n            T cells from PLWH on suppressive ART, the use of the coreceptor CXCR4 was prevalent among viruses amplified from naive and central memory CD4\n            <jats:sup>+<\/jats:sup>\n            T cells. Furthermore, we found that expanded numbers of identical viral sequences were most common in the effector memory population, and these identical sequences were also found in multiple different CD4\n            <jats:sup>+<\/jats:sup>\n            T cell subsets. Our results help to shed light on how a range of CD4\n            <jats:sup>+<\/jats:sup>\n            T cell subsets come to harbor HIV DNA, which is one of the major barriers to eradicating the virus from PLWH.\n          <\/jats:p>","DOI":"10.1128\/jvi.01736-19","type":"journal-article","created":{"date-parts":[[2019,12,16]],"date-time":"2019-12-16T12:57:46Z","timestamp":1576501066000},"update-policy":"https:\/\/doi.org\/10.1128\/asmj-crossmark-policy-page","source":"Crossref","is-referenced-by-count":27,"title":["CXCR4-Using HIV Strains Predominate in Naive and Central Memory CD4\n            <sup>+<\/sup>\n            T Cells in People Living with HIV on Antiretroviral Therapy: Implications for How Latency Is Established and Maintained"],"prefix":"10.1128","volume":"94","author":[{"given":"Michael","family":"Roche","sequence":"first","affiliation":[{"name":"The Peter Doherty Institute for Infection and Immunity, University of Melbourne and Royal Melbourne Hospital, Melbourne, 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