{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2023,1,10]],"date-time":"2023-01-10T16:15:43Z","timestamp":1673367343128},"reference-count":36,"publisher":"American Society for Microbiology","issue":"22","license":[{"start":{"date-parts":[[2007,11,15]],"date-time":"2007-11-15T00:00:00Z","timestamp":1195084800000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/journals.asm.org\/non-commercial-tdm-license"}],"content-domain":{"domain":["journals.asm.org"],"crossmark-restriction":true},"short-container-title":["J Bacteriol"],"published-print":{"date-parts":[[2007,11,15]]},"abstract":"<jats:title>ABSTRACT<\/jats:title>\n          <jats:p>\n            In\n            <jats:italic>Bacillus subtilis<\/jats:italic>\n            , the synthesis of enzymes involved in the degradation of arabinose-containing polysaccharides is subject to carbon catabolite repression (CCR). Here we show that CcpA is the major regulator of repression of the arabinases genes in the presence of glucose. CcpA acts via binding to one\n            <jats:italic>cre<\/jats:italic>\n            each in the promoter regions of the\n            <jats:italic>abnA<\/jats:italic>\n            and\n            <jats:italic>xsa<\/jats:italic>\n            genes and to two\n            <jats:italic>cre<\/jats:italic>\n            s in the\n            <jats:italic>araABDLMNPQ-abfA<\/jats:italic>\n            operon. The contributions of the coeffectors HPr and Crh to CCR differ according to growth phase. HPr dependency occurs during both exponential growth and the transitional phase, while Crh dependency is detected mainly at the transitional phase. Our results suggest that Crh synthesis may increase at the end of exponential growth and consequently contribute to this effect, together with other factors.\n          <\/jats:p>","DOI":"10.1128\/jb.01217-07","type":"journal-article","created":{"date-parts":[[2007,9,8]],"date-time":"2007-09-08T00:40:10Z","timestamp":1189212010000},"page":"8371-8376","update-policy":"http:\/\/dx.doi.org\/10.1128\/asmj-crossmark-policy-page","source":"Crossref","is-referenced-by-count":7,"title":["<i>trans<\/i>\n            -Acting Factors and\n            <i>cis<\/i>\n            Elements Involved in Glucose Repression of Arabinan Degradation in\n            <i>Bacillus subtilis<\/i>"],"prefix":"10.1128","volume":"189","author":[{"given":"Jose\u0301 Manuel","family":"Ina\u0301cio","sequence":"first","affiliation":[{"name":"Instituto de Tecnologia Qui\u0301mica e Biolo\u0301gica, Universidade Nova de Lisboa, Avenida da Repu\u0301blica, Apartado 127, 2781-901 Oeiras, Portugal"}]},{"given":"Isabel","family":"de Sa\u0301-Nogueira","sequence":"additional","affiliation":[{"name":"Instituto de Tecnologia Qui\u0301mica e Biolo\u0301gica, Universidade Nova de Lisboa, Avenida da Repu\u0301blica, Apartado 127, 2781-901 Oeiras, Portugal"},{"name":"Faculdade de Cie\u0302ncias e Tecnologia, Universidade Nova de Lisboa, Quinta da Torre, 2829-516 Caparica, Portugal"}]}],"member":"235","reference":[{"key":"e_1_3_2_2_2","doi-asserted-by":"crossref","first-page":"1","DOI":"10.1016\/S1874-5261(97)80003-0","volume":"1","year":"1997","unstructured":"Beldman, G., H. A. Schols, S. M. Pitson, M. J. F. Searl-van Leeuwen, and A. G. Voragen. 1997. Arabinans and arabinan degrading enzymes. Adv. Macromol. Carbohydr. Res. 1 : 1-64.","journal-title":"Adv. Macromol. Carbohydr. Res."},{"key":"e_1_3_2_3_2","doi-asserted-by":"crossref","first-page":"133","DOI":"10.1016\/S1096-7176(03)00009-0","volume":"5","year":"2003","unstructured":"Blencke, H.-M., G. Homuth, H. Ludwig, U. Mader, M. Hecker, and J. St\u00fclke. 2003. Transcriptional profiling of gene expression in response to glucose in Bacillus subtilis: regulation of the central metabolic pathways. Metab. Eng. 5 : 133-149.","journal-title":"Metab. Eng."},{"key":"e_1_3_2_4_2","doi-asserted-by":"crossref","first-page":"141","DOI":"10.1111\/j.1574-6968.2002.tb11123.x","volume":"209","year":"2002","unstructured":"Br\u00fcckner, R., and F. Titgemeyer. 2002. Carbon catabolite repression in bacteria: choice of carbon source and autoregulatory limitation of sugar utilization. FEMS Microbiol. 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