{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2022,4,1]],"date-time":"2022-04-01T08:19:58Z","timestamp":1648801198797},"reference-count":4,"publisher":"BMJ","issue":"11","content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Archives of Disease in Childhood"],"published-print":{"date-parts":[[1975,11,1]]},"DOI":"10.1136\/adc.50.11.907","type":"journal-article","created":{"date-parts":[[2008,12,22]],"date-time":"2008-12-22T21:14:24Z","timestamp":1229980464000},"page":"907-907","source":"Crossref","is-referenced-by-count":0,"title":["Letter: Infantile hypertrophic pyloric stenosis."],"prefix":"10.1136","volume":"50","author":[{"given":"W D","family":"Cochran","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"239","reference":[{"key":"ref_1","first-page":"151","article-title":"Unconjugated hyperbilirubinaemia in neonates with congenital gastrointestinal obstruction","volume":"64","author":"Felsher, B.F.; Asch, M.; Carpio, N.; Wooley, M.","year":"1973","unstructured":"Felsher, B. F., Asch, M., Carpio, N., and Wooley, M. (1973). Unconjugated hyperbilirubinaemia in neonates with congenital gastrointestinal obstruction. (Asbt.) Gastroenterology, 64, 151.","journal-title":"(Asbt.) Gastroenterology"},{"key":"ref_2","doi-asserted-by":"crossref","first-page":"299","DOI":"10.1172\/JCI101304","article-title":"Latent liver disease in persons recovered from catarrhal jaundice and in otherwise normal medical students as revealed by the bilirubin excretion test","volume":"21","author":"Kornberg, A.","year":"1942","unstructured":"Kornberg, A. (1942). Latent liver disease in persons recovered from catarrhal jaundice and in otherwise normal medical students as revealed by the bilirubin excretion test. Journal of Clinical Investigation, 21, 299.","journal-title":"Journal of Clinical Investigation"},{"key":"ref_3","article-title":"Dodge comments as follows: I was interested to read Dr. Cochran's kind comments on my article, and in particular his views on the occurrence of jaundice in infantile pyloric stenosis, and apologize for overlooking the possibility that alkalosis may be a contributory factor. There is no doubt that correction of metabolic ababnormalities is an important factor in the resolution of jaundice in these babies, but there is no good evidence that it is the restoration of fluid or of acid-base balance which effects the response. Thus, in the only one of my 12 jaundiced patients with really adequate biochemical documentation, there was no clinical evidence of dehydration and his plasma bicarbonate was 24 mEq\/l on admission and rose to 30-6 mEq\/l on the day of operation (3 days later), by which time the jaundice was already fading. I remember attending a C. P. C. in Boston on 29 November 1961, when Dr. Cochran himself discussed a jaundiced 'pyloric'. The patient's bicarbonate level was recorded as 24-8 mEq\/l","author":"Dr","unstructured":"Dr. Dodge comments as follows: I was interested to read Dr. Cochran's kind comments on my article, and in particular his views on the occurrence of jaundice in infantile pyloric stenosis, and apologize for overlooking the possibility that alkalosis may be a contributory factor. There is no doubt that correction of metabolic ababnormalities is an important factor in the resolution of jaundice in these babies, but there is no good evidence that it is the restoration of fluid or of acid-base balance which effects the response. Thus, in the only one of my 12 jaundiced patients with really adequate biochemical documentation, there was no clinical evidence of dehydration and his plasma bicarbonate was 24 mEq\/l on admission and rose to 30-6 mEq\/l on the day of operation (3 days later), by which time the jaundice was already fading. I remember attending a C.P.C. in Boston on 29 November 1961, when Dr. Cochran himself discussed a jaundiced 'pyloric'. The patient's bicarbonate level was recorded as 24-8 mEq\/l."},{"key":"ref_4","article-title":"It seems to me that the most likely explanation for jaundice is impairment of hepatic glucuronyl transferase activity by reduced caloric intake. This hypothesis has been studied by Felsher","year":"1973","unstructured":"It seems to me that the most likely explanation for jaundice is impairment of hepatic glucuronyl transferase activity by reduced caloric intake. This hypothesis has been studied by Felsher et al. (1973), who found depressed activity of the UDPG-transferase system in surgical liver biopsies from jaundiced infants with gastrointestinal obstruction, but a tenfold higher level in nonicteric infants with similar disorders. As I pointed out in my article, the association of jaundice with","journal-title":"et al; who found depressed activity of the UDPG-transferase system in surgical liver biopsies from jaundiced infants with gastrointestinal obstruction, but a tenfold higher level in nonicteric infants with similar disorders"}],"container-title":["Archives of Disease in Childhood"],"original-title":[],"language":"en","link":[{"URL":"https:\/\/syndication.highwire.org\/content\/doi\/10.1136\/adc.50.11.907","content-type":"unspecified","content-version":"vor","intended-application":"similarity-checking"}],"deposited":{"date-parts":[[2020,9,26]],"date-time":"2020-09-26T09:03:59Z","timestamp":1601111039000},"score":1,"resource":{"primary":{"URL":"https:\/\/adc.bmj.com\/lookup\/doi\/10.1136\/adc.50.11.907"}},"subtitle":[],"short-title":[],"issued":{"date-parts":[[1975,11,1]]},"references-count":4,"journal-issue":{"issue":"11","published-print":{"date-parts":[[1975,11,1]]}},"URL":"https:\/\/doi.org\/10.1136\/adc.50.11.907","relation":{},"ISSN":["0003-9888","1468-2044"],"issn-type":[{"value":"0003-9888","type":"print"},{"value":"1468-2044","type":"electronic"}],"subject":[],"published":{"date-parts":[[1975,11,1]]}}}