{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2022,11,28]],"date-time":"2022-11-28T20:08:43Z","timestamp":1669666123007},"reference-count":11,"publisher":"BMJ","issue":"4","content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Archives of Disease in Childhood"],"published-print":{"date-parts":[[1976,4,1]]},"DOI":"10.1136\/adc.51.4.314","type":"journal-article","created":{"date-parts":[[2008,12,22]],"date-time":"2008-12-22T21:14:24Z","timestamp":1229980464000},"page":"314-316","source":"Crossref","is-referenced-by-count":4,"title":["Thyrotoxicosis in a neonate of a mother with no history of thyroid disease."],"prefix":"10.1136","volume":"51","author":[{"given":"D S","family":"Brookfield","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"A E","family":"McCandless","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"C S","family":"Smith","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"239","reference":[{"key":"ref_1","doi-asserted-by":"crossref","first-page":"699","DOI":"10.1210\/jcem-18-7-699","article-title":"The presence of an abnormal thyroid stimulating hormone in the serum of some thyrotoxic patients","volume":"18","author":"Adams, D.D.","year":"1958","unstructured":"Adams, D. D. (1958). The presence of an abnormal thyroid stimulating hormone in the serum of some thyrotoxic patients. Journal of Clinical Endocrinology and Metabolism, 18, 699.","journal-title":"Journal of Clinical Endocrinology and Metabolism"},{"key":"ref_2","first-page":"446","article-title":"Hereditary aspects of Graves'","volume-title":"disease in infancy and childhood. J7ournal of Pediatrics","volume":"81","author":"Hollingsworth, D.R.; Mabry, C.C.; Eckerd, J.M.","year":"1972","unstructured":"Hollingsworth, D. R., Mabry, C. C., and Eckerd, J. M. (1972). Hereditary aspects of Graves' disease in infancy and childhood. J7ournal of Pediatrics, 81, 446."},{"key":"ref_3","doi-asserted-by":"crossref","first-page":"660","DOI":"10.1210\/jcem-24-7-660","article-title":"Neonatal Graves'","volume":"24","author":"McKenzie, J.M.","year":"1964","unstructured":"McKenzie, J. M. (1964). Neonatal Graves' disease. Journal of Clinical Endocrinology and Metabolism, 24, 660.","journal-title":"disease. Journal of Clinical Endocrinology and Metabolism"},{"key":"ref_4","doi-asserted-by":"crossref","first-page":"695","DOI":"10.1136\/bmj.4.5946.695","article-title":"Neonatal hyperthyroidism and long-acting thyroid-stimulator protector","volume":"4","author":"Nutt, J.; Clark, F.; Welch, R.G.; Hall, R.","year":"1974","unstructured":"Nutt, J., Clark, F., Welch, R. G., and Hall, R. (1974). Neonatal hyperthyroidism and long-acting thyroid-stimulator protector. British Medical Journal, 4, 695.","journal-title":"British Medical Journal"},{"key":"ref_5","doi-asserted-by":"crossref","first-page":"916","DOI":"10.1016\/S0022-3476(72)80543-2","article-title":"Thyroid function assays in infants","volume":"81","author":"O'Halloran, M.T.; Webster, H.L.","year":"1972","unstructured":"O'Halloran, M. T., and Webster, H. L. (1972). Thyroid function assays in infants. Journal of Pediatrics, 81, 916.","journal-title":"Journal of Pediatrics"},{"key":"ref_6","doi-asserted-by":"crossref","first-page":"292","DOI":"10.1056\/NEJM196302072680606","article-title":"Neonatal hyperthyroidism","volume":"268","author":"Rosenberg, D.; Grand, M.J.H.; Silbert, D.","year":"1963","unstructured":"Rosenberg, D., Grand, M. J. H., and Silbert, D. (1963). Neonatal hyperthyroidism. New England Journal of Medicine, 268, 292.","journal-title":"New England Journal of Medicine"},{"key":"ref_7","doi-asserted-by":"crossref","first-page":"1024","DOI":"10.1016\/S0140-6736(69)90638-2","article-title":"Long-acting thyroid stimulator in euthyroid relatives of thyrotoxic patients","volume":"2","author":"Wall, J.R.; Good, B.F.; Hetzel, B.S.","year":"1969","unstructured":"Wall, J. R., Good, B. F., and Hetzel, B. S. (1969). Long-acting thyroid stimulator in euthyroid relatives of thyrotoxic patients. Lancet, 2, 1024.","journal-title":"Lancet"},{"key":"ref_8","article-title":"Neonatal brain growth during prolonged intravenous feeding Total intravenous feeding may occasionally be a life-saving procedure for the seriously ill low birthweight infant. The technical and metabolic complications associated with total intravenous nutrition have been well reviewed (Harries","volume-title":"Baum and Aynsley-Green","author":"Harries, 1972, Heird","year":"1971","unstructured":"Neonatal brain growth during prolonged intravenous feeding Total intravenous feeding may occasionally be a life-saving procedure for the seriously ill low birthweight infant. The technical and metabolic complications associated with total intravenous nutrition have been well reviewed (Harries, 1971; Harries, 1972; Heird et al., 1972; Heird and Winters, 1975; Baum and Aynsley-Green, 1975). However, little is known about the effects of prolonged intravenous feeding on tissue growth and development in the newborn. In particular it would be important to know the effects of longterm intravenous nutrition on the brain, since in the neonatal period when the brain is growing rapidly it may be susceptible to nutritional deprivation (Dobbing, 1974)."},{"key":"ref_9","article-title":"have provided standards for the biochemical development of the neonatal brain by analysing brains ranging in age from 10 weeks' gestation to 7 postnatal years. Using these standards for comparison, we have attempted to assess the effects of long-term intravenous nutrition on the growth and development of the brain from an infant who was born at 32 weeks' gestation and who died after 10 weeks of continuous intravenous feeding","author":"Dobbing; Sands","year":"1973","unstructured":"Dobbing and Sands (1973) have provided standards for the biochemical development of the neonatal brain by analysing brains ranging in age from 10 weeks' gestation to 7 postnatal years. Using these standards for comparison, we have attempted to assess the effects of long-term intravenous nutrition on the growth and development of the brain from an infant who was born at 32 weeks' gestation and who died after 10 weeks of continuous intravenous feeding."},{"key":"ref_10","article-title":"Details of the intravenous feeds which this infant received are given in Table I. For most of his life he received 100 cal\/kg per 24 hours, 4 g of fat as intralipid\/kg per 24 hours, and 3 g amino acids\/kg per 24 hours. In addition, attempts were made to maintain homoeostasis for electrolytes, vitamins, and trace minerals. The most serious metabolic problem documented was hypophosphataemia, which was corrected by the addition of increased phosphate supplements and has been reported elsewhere","year":"1974","unstructured":"Details of the intravenous feeds which this infant received are given in Table I. For most of his life he received 100 cal\/kg per 24 hours, 4 g of fat as intralipid\/kg per 24 hours, and 3 g amino acids\/kg per 24 hours. In addition, attempts were made to maintain homoeostasis for electrolytes, vitamins, and trace minerals. The most serious metabolic problem documented was hypophosphataemia, which was corrected by the addition of increased phosphate supplements and has been reported elsewhere (Woods et al., 1974)."},{"key":"ref_11","article-title":"Growth on intravenous feeding. The infant received continuous intravenous feeding from day 5 after birth until death 10 weeks later, during which time there was a strong clinical impression of increase in length and, to a limited extent, in muscle bulk. Unfortunately no clinical measurements of length nor of limb girth were made. Birthweight was 1270 g, which is on the 10th centile for 32 weeks, and at necropsy weight was 1680 g, below the 3rd centile for 42 weeks' gestation. The occipitofrontal head circumference at birth was 29 cm, on the 50th centile for 32 weeks, whereas the post-mortem head circumference was 33 cm, 10th centile for 42 weeks'","year":"1972","unstructured":"Growth on intravenous feeding. The infant received continuous intravenous feeding from day 5 after birth until death 10 weeks later, during which time there was a strong clinical impression of increase in length and, to a limited extent, in muscle bulk. Unfortunately no clinical measurements of length nor of limb girth were made. Birthweight was 1270 g, which is on the 10th centile for 32 weeks, and at necropsy weight was 1680 g, below the 3rd centile for 42 weeks' gestation. The occipitofrontal head circumference at birth was 29 cm, on the 50th centile for 32 weeks, whereas the post-mortem head circumference was 33 cm, 10th centile for 42 weeks' gestation (standards from Davies et al., 1972)."}],"container-title":["Archives of Disease in Childhood"],"original-title":[],"language":"en","link":[{"URL":"https:\/\/syndication.highwire.org\/content\/doi\/10.1136\/adc.51.4.314","content-type":"unspecified","content-version":"vor","intended-application":"similarity-checking"}],"deposited":{"date-parts":[[2020,9,26]],"date-time":"2020-09-26T09:06:39Z","timestamp":1601111199000},"score":1,"resource":{"primary":{"URL":"https:\/\/adc.bmj.com\/lookup\/doi\/10.1136\/adc.51.4.314"}},"subtitle":[],"short-title":[],"issued":{"date-parts":[[1976,4,1]]},"references-count":11,"journal-issue":{"issue":"4","published-print":{"date-parts":[[1976,4,1]]}},"URL":"https:\/\/doi.org\/10.1136\/adc.51.4.314","relation":{},"ISSN":["0003-9888","1468-2044"],"issn-type":[{"value":"0003-9888","type":"print"},{"value":"1468-2044","type":"electronic"}],"subject":[],"published":{"date-parts":[[1976,4,1]]}}}