{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,6,22]],"date-time":"2026-06-22T16:42:52Z","timestamp":1782146572884,"version":"3.54.5"},"reference-count":13,"publisher":"BMJ","content-domain":{"domain":["bmj.com"],"crossmark-restriction":true},"short-container-title":["BMJ Case Reports"],"accepted":{"date-parts":[[2018,9,16]]},"abstract":"<jats:p>Posterior urethral valves (PUV) are an important cause of paediatric obstructive uropathy. PUV are usually diagnosed by prenatal ultrasonography (US) revealing hydronephrosis and bladder distention. We describe a 17-day-old male infant with abdominal distention who had no hydronephrosis on prenatal US. Laboratory investigations showed serum creatinine of 12\u2009mg\/dL, hyperkalaemia and metabolic acidosis. Abdominal US showed large amount of ascites, normal-sized kidneys without hydronephrosis and incompletely distended bladder. Paracentesis revealed clear, yellow ascitic fluid with creatinine level of 27\u2009mg\/dL compatible with urinary ascites. Voiding cystourethrogram (VCUG) demonstrated PUV with a dilated posterior urethra, grade 5 right vesicoureteral reflux and a ruptured kidney fornix with peritoneal extravasation of contrast. Foley decompression resulted in normalisation of creatinine within 72\u2009hours. Transurethral resection of PUV was performed, and a repeat VCUG showed recovery of forniceal rupture. This case illustrates an unusual presentation of a potentially life-threatening but treatable cause of urinary tract obstruction.<\/jats:p>","DOI":"10.1136\/bcr-2018-225053","type":"journal-article","created":{"date-parts":[[2018,10,13]],"date-time":"2018-10-13T01:45:20Z","timestamp":1539395120000},"page":"bcr-2018-225053","update-policy":"https:\/\/doi.org\/10.1136\/crossmarkpolicy","source":"Crossref","is-referenced-by-count":2,"title":["Neonate with urinary ascites but no hydronephrosis: unusual presentation of posterior urethral valves"],"prefix":"10.1136","volume":"2018","author":[{"given":"Elisabetta","family":"Prat","sequence":"first","affiliation":[{"name":"Pediatrics, Inova Children\u2019s Hospital, Falls Church, Virginia, USA"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Patricia","family":"Seo-Mayer","sequence":"additional","affiliation":[{"name":"Pediatric Nephrology, Pediatric Specialists of Virginia, Fairfax, Virginia, USA"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Swati","family":"Agarwal","sequence":"additional","affiliation":[{"name":"Pediatric Critical Care, Inova Children\u2019s Hospital, Falls Church, Virginia, USA"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"239","published-online":{"date-parts":[[2018,10,12]]},"reference":[{"key":"2025082723421845000_2018.oct12_1.bcr-2018-225053.1","doi-asserted-by":"publisher","DOI":"10.1007\/s12519-011-0289-1"},{"key":"2025082723421845000_2018.oct12_1.bcr-2018-225053.2","doi-asserted-by":"crossref","first-page":"217","DOI":"10.1055\/s-2007-965148","article-title":"Fetal urinary bladder rupture and urinary ascites secondary to posterior urethral valves. 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