{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2025,11,16]],"date-time":"2025-11-16T20:21:10Z","timestamp":1763324470035,"version":"3.45.0"},"reference-count":0,"publisher":"BMJ","issue":"5699","license":[{"start":{"date-parts":[[1970,3,28]],"date-time":"1970-03-28T00:00:00Z","timestamp":7430400000},"content-version":"tdm","delay-in-days":0,"URL":"http:\/\/www.bmj.com\/company\/legal-information\/terms-conditions\/legal-information\/tdm-licencepolicy"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Br Med J"],"published-print":{"date-parts":[[1970,3,28]]},"abstract":"<jats:p>Serial measurements of total body potassium in 21 patients with chronic renal failure being treated with three 10-hour periods of dialysis per week, against a dialysate fluid containing 1\u00b75 mEq of potassium per litre, showed no evidence of potassium depletion. Mild hyperkalaemia was found in some patients before dialysis, correlated with the pre-dialysis hydrogen ion concentration. Hypokalaemia occurred during dialysis in almost half of the studies made; the plasma potassium concentration, however, rose to normal levels within two to four hours of stopping dialysis. A delay in the movement of potassium from the cells into the extracellular fluid is suggested as a cause for the observed hypokalaemia.<\/jats:p>\n                  <jats:p>\n                    In all but one patient the pre-dialysis blood\n                    <jats:italic>p<\/jats:italic>\n                    H was normal, but rose to alkalaemic levels during dialysis. A pronounced degree of hypocapnia was noted before dialysis, and this was not altered by a rising blood\n                    <jats:italic>p<\/jats:italic>\n                    H during dialysis. It is suggested that a stimulus to respiration other than the hydrogen ion gradient between the brain cells and cerebral spinal fluid may produce the observed hypocapnia.\n                  <\/jats:p>","DOI":"10.1136\/bmj.1.5699.779","type":"journal-article","created":{"date-parts":[[2009,4,2]],"date-time":"2009-04-02T07:02:10Z","timestamp":1238655730000},"page":"779-783","source":"Crossref","is-referenced-by-count":21,"title":["Potassium Balance and Acid-Base Changes in Patients Undergoing Regular Haemodialysis Therapy"],"prefix":"10.1136","volume":"1","author":[{"given":"A. G.","family":"Morgan","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"L.","family":"Burkinshaw","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"P. J. A.","family":"Robinson","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"S. M.","family":"Rosen","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"239","published-online":{"date-parts":[[1970,3,28]]},"container-title":["BMJ"],"original-title":[],"language":"en","link":[{"URL":"http:\/\/data.bmj.org\/tdm\/10.1136\/bmj.1.5699.779","content-type":"unspecified","content-version":"vor","intended-application":"text-mining"},{"URL":"https:\/\/syndication.highwire.org\/content\/doi\/10.1136\/bmj.1.5699.779","content-type":"unspecified","content-version":"vor","intended-application":"similarity-checking"}],"deposited":{"date-parts":[[2025,11,16]],"date-time":"2025-11-16T20:16:28Z","timestamp":1763324188000},"score":1,"resource":{"primary":{"URL":"https:\/\/www.bmj.com\/lookup\/doi\/10.1136\/bmj.1.5699.779"}},"subtitle":[],"short-title":[],"issued":{"date-parts":[[1970,3,28]]},"references-count":0,"journal-issue":{"issue":"5699","published-online":{"date-parts":[[1970,3,28]]},"published-print":{"date-parts":[[1970,3,28]]}},"alternative-id":["10.1136\/bmj.1.5699.779"],"URL":"https:\/\/doi.org\/10.1136\/bmj.1.5699.779","relation":{},"ISSN":["0007-1447","1468-5833"],"issn-type":[{"type":"print","value":"0007-1447"},{"type":"electronic","value":"1468-5833"}],"subject":[],"published":{"date-parts":[[1970,3,28]]}}}